Thread: Post PCT Cruise hmmmmm

Originally Posted by OnPoint88

I'm on my pre pct hcg now and not looking forward to weeks on Nolva and Clomid because it makes me feel like I'm shooting estrogen, so I was brainstorming on how to fill the void. I was thinking Primo and Var have minimal affect on the HPTA and Proviron apparently has a postive affect on LH so I'm think about stacking these post pct after some blood work showing everything is normal and then seeing if it stays normal on this little experiment. I might even be able to keep it in check with 250mcg of Letro which I have a ton of and maybe some finasteride which also as shown to have a positive affect on Test while reducing the affects of dht so that would be great for my hair and prostate. Considering Prostate cancer is one of the top killers in men I'm starting to think that finasteride should be mandatory on any cycle. I'm sure our sponsors would love to see a ton of people on this kind of cruise lol it's a win win situation and hopefully my labs will back it up.

Primo, Var and Proviron are anabolic steroids and WILL suppress your HPTA, even in small doses.

Originally Posted by GMO

Primo, Var and Proviron are anabolic steroids and WILL suppress your HPTA, even in small doses.

^^this. I'd like to know who started all this bs about guys running d Bol or var for "pct". If you take any synthetic hormones you are not post cycle you are still on cycle. If you don't want to do pct don't fuck around with something dangerous like hand fulls of orals. Cruise on test get blood work and train hard. You are either "on" or "off" period there's no grey area.

Acta Endocrinol (Copenh). 1968 Nov;59(3):497-507.
Androgen treatment without inhibiting effect on hypophysis and male gonads. Biochemical and morphological studies after treatment with Mesterolone.

Petry R, Rausch-Stroomann JG, Hienz HA, Senge T, Mauss J.
PMID:4880965[PubMed - indexed for MEDLINE]


Arzneimittelforschung. 1970 Apr;20(4):545-7.
Comparative studies about the influence of metenoloneacetate and mesterolone on hypophysis and male gonads.

Petry R, Trenkner G, Senge T, Hienz HA, Mauss J, Rausch-Stroomann JG.
PMID:5467820 [PubMed - indexed for MEDLINE

Clin Endocrinol (Oxf). 1993 Apr;38(4):393-8.
The effects of oxandrolone on the growth hormone and gonadal axes in boys with constitutional delay of growth and puberty.
Malhotra A, Poon E, Tse WY, Pringle PJ, Hindmarsh PC, Brook CG.

Endocrine Unit, Middlesex Hospital, London, UK.

OBJECTIVE: We studied the effects of oxandrolone on serum concentrations of LH, FSH, testosterone, GH, SHBG, DHEAS, IGF-I and insulin in boys with constitutional delay of growth and puberty. DESIGN: Ten boys with constitutional delay of growth and puberty, mean age 13.8 years (range 12.4-15.5) were studied. Twenty-four-hour serum concentration profiles of GH, LH and FSH were constructed by drawing blood samples at 20-minute intervals. Three study occasions over a period of 6 months were chosen to assess hormone concentrations before, during and 6 weeks after a 3-month course of oxandrolone (2.5 mg once daily) therapy. RESULTS: Growth velocity increased during oxandrolone treatment and stayed higher after therapy (pre 3.9 +/- 0.5; on 6.3 +/- 0.8; post 6.4 +/- 0.9 cm/year (mean +/- SEM) two way ANOVA, F = 5.3, P = 0.02). Oxandrolone had androgenic effects, suppressing mean serum LH concentrations from 1.7 +/- 0.3 to 1.1 +/- 0.2 U/I and serum testosterone concentrations from 1.9 +/- 0.6 to 0.8 +/- 0.1 nmol/l. SHBG concentrations were also reduced from 130.9 +/- 14.6 to 30.7 +/- 7.3 nmol/l. Serum GH concentration fell slightly from 5.9 +/- 0.6 to 4.8 +/- 0.5 mU/l. After cessation of treatment, there was a significant 'rebound' in mean 24-hour serum LH (2.6 U/l +/- 0.4) and testosterone concentrations (3.2 +/- 0.9 nmol/l) but no change in serum GH concentrations. SHBG values also rose but not to the same extent as those observed before therapy (82.0 +/- 8.4 nmol/l). There were no statistically significant differences in serum concentrations of FSH, DHEAS, IGF-I and insulin over the study period. In a stepwise multiple regression analysis of factors that might influence the growth rate observed, the 24-hour mean serum testosterone concentration and the treatment (on or off) with oxandrolone were the main influences. The relationship was described by the equation Height velocity = 0.69 (24-hour mean serum testosterone concentration)+1.70 (treatment regimen)+3.37 (adjusted R2 = 0.35, F = 8.39, P = 0.001). CONCLUSIONS: Oxandrolone has an androgenic action as shown by changes in serum LH, testosterone and SHBG concentrations and by the lack of effect on FSH. No effect of oxandrolone on the GH axis was documented. We suggest that the growth promoting effects of oxandrolone are related in part to the mild androgenic effects of the steroid and the growth acceleration following oxandrolone withdrawal may reflect increasing total serum testosterone concentrations and decreasing levels of SHBG and progress in puberty.

PMID: 8319371 [PubMed - indexed for MEDLINE]

There's no need to do a PCT if you decide to do that. It's called crusing.