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Originally posted by Dale Mabry We all know that we will generally lose some muscle postcycle. We also know that T3 eats muscle and fat indiscriminately. So what if we decided to take T3 towards the end of a cycle which should, in turn, temporarily reduce our own thyroid production during postcycle? Asssuming we were getting enough cals during the end of the cycle, we would have increased protein utilization followed by a period of low thyroid output during PCT. Would this help limit the muscle loss found postcycle? If we could control cortisol with Phosphatidylserine, could this be the magic bullet we are looking for to limit muscle loss postcycle? Whatcha all think? Prolly would not be the most productive PCT weeks of our lives but, assuming you could handle the crash, would this retain some LBM until we get our endogenous test levels back up? |