Thread: Viagra used for lifting

No. It was (and is) a heart medication before it was used for E.D. It's an N.O. donor - basically, NOxplode that actually WORKS.

It also appears to stimulate an increase in test production in males.

I believe some organizations are banning the use of viagra because of the benefits it has on training and exercise.

lol never heard of that one before

Originally Posted by misllika

HI
People inflicted by asthma suffer from symptoms such as repeated episodes of cough, chest congestion, wheezing, sinus irritation, and shortness of breaths. This chronic illness is characterized by inflammation, swelling, and constriction of the air passage and the increased production of mucous in the passage. Often triggered by viral illnesses (eg, the common cold), pollen, foods, intense exercise or environmental conditions

... and what does this have to do with this thread?

Originally Posted by Built

No. It was (and is) a heart medication before it was used for E.D. It's an N.O. donor - basically, NOxplode that actually WORKS.

It also appears to stimulate an increase in test production in males.

Believe it or not(and thisis how you know that somethig is legit or not due to gambling) it is banned in horseracing.It is a legit NO booster that has been proven to effect the outcome of races.

SIGN me up!!!

Players in the MLB use this stuff all the time for working out.. its been in the paper a few times & how it could be a unfair advantage for some over the others that do not take it. Last I read there were a few dicks out there that were trying to actually bann it from MLB lol

Hmm interesting...
Viagra Improves High Altitude Exercise Performance Up To 45% For Some

This is what I need! Ha NOT, well maybe...

I'll have to dig and see if Cialis works the same.

Hmmm. Looks like it does: The Type 5 Phosphodiesterase Inhibitor Tadalafil Influences Salivary Cortisol, Testosterone, and Dehydroepiandrosterone Sulphate Responses to Maximal Exercise in Healthy Men -- Di Luigi et al. 93 (9): 3510 -- Journal of Clinical Endocrinology & Metab


The Type 5 Phosphodiesterase Inhibitor Tadalafil Influences Salivary Cortisol, Testosterone, and Dehydroepiandrosterone Sulphate Responses to Maximal Exercise in Healthy Men
Luigi Di Luigi, Carlo Baldari, Paolo Sgrò, Gian Pietro Emerenziani, Maria Chiara Gallotta, Serena Bianchini, Francesco Romanelli, Fabio Pigozzi, Andrea Lenzi and Laura Guidetti

Units of Endocrinology (L.D.L., P.S., S.B.), Sport Sciences (C.B., G.P.E.), Exercise Sciences (M.C.G., L.G.), and Sport Medicine (F.P.), Department of Health Sciences, University of Rome "Foro Italico," 00194 Rome, Italy; and Department of Medical Physiopathology (F.R., A.L.), University of Rome "La Sapienza," 00161 Rome, Italy

Address all correspondence and requests for reprints to: Professor Luigi Di Luigi, M.D., Unit of Endocrinology, Department of Health Sciences, University of Rome "Foro Italico," Piazza Lauro de Bosis, 15, 00194 Rome, Italy. E-mail: luigi.diluigi@iusm.it.

Context: Physical exercise-related stress activates hypothalamus-pituitary-adrenal (HPA) axis; nitric oxide is one of the mediators of the HPA axis response to stress, and phosphodiesterase type 5 inhibitors influences nitric oxide-linked biological activities.

Objective: The objective of the study was to investigate whether a single oral long half-life phosphodiesterase type 5 inhibitor (tadalafil) administration influences the HPA axis response to exercise-related stress.

Design: This was a double-blind, cross-over trial.

Participants: Participants included nine healthy male athletes.

Interventions: All subjects performed a maximal exercise test in normoxia, after which they received a single oral administration of tadalafil or placebo. Then after a 2-wk washout period, they were crossed over and repeated the exercise test. Each subject was his own control. Salivary collections, for steroid evaluations [cortisol, dehydroepiandrosterone sulphate (DHEAS), testosterone] and respective ratio calculation (DHEAS to cortisol, testosterone to cortisol, testosterone to DHEAS), were performed before each exercise (Pre-Ex), immediately after (Post-Ex), and at 30 min during recovery.

Results: As expected, mean salivary cortisol concentration increased immediately after exercise after both tadalafil and placebo (P = 0.014 and P =0.036 vs. Pre-Ex, respectively); however, the cortisol increase was significantly higher after tadalafil administration (P = 0.034 vs. placebo). Furthermore, an increased salivary testosterone after exercise was observed only after tadalafil administration (P = 0.029 vs. Pre-Ex). No effects of either exercise and/or tadalafil administration on salivary DHEAS concentrations were observed. DHEAS to cortisol and testosterone to cortisol ratios significantly decreased after exercise after tadalafil administration (P = 0.037, and P = 0.02 vs. placebo, respectively).

Conclusion: Tadalafil administration amplified the salivary cortisol and testosterone responses to a maximal exercise-related stress in healthy trained humans.

So I can be blind with a woody and a bit of muscle, hmmm. That might be ok.