IronMagLabs


Will Green Tea Extract help for this?

Results 1 to 7 of 7
  1. #1
    Registered User
    Rocky_B's Avatar


    Join Date
    Jul 2004
    Location
    Canada
    Posts
    446
    Rep Points
    10

    Will Green Tea Extract help for this?






    Right now, for the first time in my life I can see an outline of my abs, but they aren't as defined as I'd like. I'm doing weighted and non-weighted ab exercises frequently throughout the week while at the gym and at home, and my diet is in check, and I run 3.5 kilometres about 2-3 times a week, in addition to training with weights in the gym 5 days a week. I take Whey, just started creatine for the first time less than a week ago (not loading), multis and fish oils, but haven't tried green tea extract. I'm lean but will green tea extract help in defining my abs further?
    IMA BEAST

  2. #2
    Senior Member
    ADMINISTRATOR
    Prince's Avatar


    Join Date
    Nov 2000
    Gender
    Male
    Location
    Panama
    Posts
    62,587
    Rep Points
    2147483647

    it may help a little, but a proper cutting diet is the key.





    IronMagLabs 15% Discount Code: Robert15



    IronMag Research 15% Discount Code: Robert15



  3. #3
    Registered User
    Rocky_B's Avatar


    Join Date
    Jul 2004
    Location
    Canada
    Posts
    446
    Rep Points
    10

    Will it help because of the caffeine content or because of the green tea? I would be buying one that had no caffeine in it, because I'd have as little caffeine as possible in my body.
    IMA BEAST

  4. #4
    Senior Member
    ADMINISTRATOR
    Prince's Avatar


    Join Date
    Nov 2000
    Gender
    Male
    Location
    Panama
    Posts
    62,587
    Rep Points
    2147483647

    Quote Originally Posted by Rocky_B
    Will it help because of the caffeine content or because of the green tea? I would be buying one that had no caffeine in it, because I'd have as little caffeine as possible in my body.

    Green Tea:

    by David Tolson

    LEAN FUEL XP


    Introduction

    Next to water, tea is presently the most widely consumed beverage in the world [1]. The three forms of tea, which are differentiated by processing method, are green, oolong, and black tea. Of these, green tea undergoes the least amount of processing, and it has been used for medicinal purposes for thousands of years. Green tea has become well known for its antioxidant, antimutagenic and anticarcinogenic effects. Other possible benefits include treatment of cardiovascular disease, diabetes, dermatological problems, obesity, and oral health problems [2]. This article will examine the constituents of green tea and the various benefits they have to offer.

    Constituents of Green Tea

    Catechins - The polyphenols are generally considered to be the most important elements of green tea, with the catechins being the most important polyphenols. Catechins found in green tea include EC, EGC, ECG, and EGCG. Of these, EGCG ((-)-epigallocatechin-3-gallate) is by far the most active by itself, but the combination of catechins can be especially effective for cancer protection [3], and EC appears to increase the incorporation of EGCG and ECG into lipid bilayers [4], emphasizing the importance of synergistic effects between the various components of green tea. Although the numbers vary, green tea is generally made up of about 10% polyphenols, 50% of which are EGCG. The green tea catechins have been studied extensively and will be the primary focus of this article.

    Pheophytins, chlorophylls, and carotenoids - Despite the importance of catechins, they are not the only constituents of green tea that offer health benefits. Some studies find that catechins depend on other components or are not even major players in some of the anti-genotoxic and antioxidant effects of green tea [5, 6]. The numerous other active compounds in green tea identified include chlorophylls a and b, pheophytins a and b, lutein, and beta-carotene. All of them have antioxidant properties [7], and the health benefits of beta-carotene and lutein (such as prevention of macular degeneration) are well known. Chorophylls and pheophytins from green tea are also anti-carcinogenic [8, 9], but a discussion of the many benefits these phytonutrients have to offer is beyond the scope of this article.

    Theanine - L-theanine is an amino acid found in high concentrations in tea, with a typical cup of tea containing 30 mg or more. Theanine decreases blood pressure [10, 11] and has been found effective in increasing the antitumor activity of cancer drugs [12, 13], but its most important properties are in the area of the brain. L-theanine bears structural similarity to glutamic acid and hence competes with it in binding to glutamate receptors, offering protection against glutamate neurotoxicity [11]. This glutamate receptor competition also provides a variety of differential effects on the brain. These include an increase in serotonin and/or dopamine in some areas of the brain, notably the striatum, hypothalamus and hippocampus [14], an increase in GABA [15], and an increase in brain alpha wave activity [15]. While beta wave activity is associated with periods of high stress, alpha waves are associated with a state of being awake, but relaxed (such as the period right before the onset of sleep). Theanine is said to promote a state of "alert relaxation" because of these differential effects [15], and it also antagonizes the stimulation and anxiogenic (anxiety promoting) effects of caffeine [11, 16] which may be responsible for the paradoxical calming effect of green tea despite the caffeine content.

    Caffeine - The amounts of caffeine in green tea are relatively low, with 20 mg in a typical cup (about half as much as a Coke). Surprisingly, caffeine can play a significant role in the anti-mutagenic effects of green tea in some cases, especially in the prevention of UV-induced skin cancer [17, 18]. The presence of caffeine in green tea will be of most importance in the upcoming discussion of thermogenesis.

    Green Tea and body composition

    Recent research has confirmed that green tea can cause weight loss through multiple pathways. In addition to being a potent appetite supressant, green tea increases thermogenesis, preferentially burning fat over protein in a similar manner to many other thermogenics. Green tea has been demonstrated to be as or more effective than some prescription weight loss medications, and may rival the ECA stack in terms of fat loss. In addition, green tea is one of the few weight loss medications that has not been associated with negative effects on the cardiovascular system (such as increased heart rate and blood pressure) or CNS side effects (such as overstimulation and irritability). More research is needed before the level of effectiveness of green tea can be more conclusively determined, but the present data shows a clear benefit.

    Green tea has been known to be a thermogenic agent for quite some time, but the thermogenesis was usually attributed to the caffeine content. It was then found in an in vitro experiment with brown adipose tissue that the thermogenic effect of green tea was "much greater than can be attributed to its caffeine content per se" [19]. Other in vitro experiments also demonstrate that green tea inhibits lipogenesis (the creation of fat) [20, 21]. In rodents, studies with both green tea and green tea powder have shown that it decreases body weight and food intake [22, 23] and inhibits lipogenesis [24].

    Two important studies have been done assessing the thermogenic effect of green tea in humans. The first was a preliminary study that compared the effects of green tea extract (containing 150 mg caffeine and 270 mg EGCG), caffeine (150 mg), and placebo on 24-hour energy expenditure. While caffeine alone increased energy expenditure by about .6% over placebo (which was not statistically significant in this study), the green tea extract increased it by 3.5%. Additionally, while the oxidation of fat contributed to 31.6% of energy expenditure in the placebo group, in contributed 41.5% in the green tea extract group, indicating that the increase in energy expenditure was due to the breakdown of fat, not protein. Measurements of urinary nitrogen excretion, which were significantly different between treatments, further supported this contention. It is also interesting to note that thermogenic response was not correlated with body mass index (BMI), implying that green tea may be equally as effective in relatively lean individuals. Finally, the increased thermogenesis was not accompanied by an increase in heart rate, which makes green tea distinct from other thermogenic drugs [25].

    The second study was a three month open trial with 70 subjects using the same dosage of the same extract as in the above study (150 mg caffeine, 375 mg total catechins, 270 mg EGCG per day). Treatment with green tea was well tolerated and associated with a body weight reduction of 4.6% and a reduction of waist circumference of 4.5% [1]. Hopefully these promising results will be followed by larger placebo-controlled studies.

    Green tea has many mechanisms of action in stimulating weight loss. The most important is probably the inhibition of catechol-O-methyl-transferase (COMT) by EGCG [1, 19, 25]. COMT is the enzyme that breaks down norepinephrine (NE), one of the body's most important lipolytic hormones. Caffeine also plays a synergistic role by inhibiting phosophdiesterases (enzymes that break down cAMP, which is further down the lipolytic pathway) [19, 25]. Although EGCG is the most responsible, some flavanoids found in small amounts in green tea such as quercetin and myricetin also inhibit COMT and may play a minor role [25].

    Secondly, green tea decreases the digestibility of dietary fat [1, 26]. The proposed mechanism of action is inhibition of both gastric and pancreatic lipase, which has been demonstrated in vitro [1]. These enzymes both play major roles in the digestion of fat, so when they are inhibited a smaller proportion of fat is absorbed and a greater proportion excreted.

    Green tea is also a potent appetite suppressant. This can be partly explained by the fact that it increases both NE and dopamine [14, 25], but further mechanisms of action have been hypothesized. Specifically, tea polyphenols have been known to elevate levels of cholecystokinin (CCK) [2], a hormone which depresses food intake [2, 22]. It is not yet known whether this plays a significant role in the action of green tea, and one of the effects of elevated CCK is an increase in pancreatic lipase, which is actually inhibited by green tea. It could be that green tea simultaneously elevates CCK and decreases pancreatic lipase, conferring the benefits of both appetite suppression and decreased fat digestibility.

    Finally, the antioxidant properties of green tea may play a role in the lipolytic effect [20-22]. One cell culture study suggested that green tea inhibited lipogenesis by increasing superoxide dismutase activity and subsequently decreasing the formation of free radicals [20], while another suggests that vitamin C from green tea plays a role in its lipolytic activity [21]. Even if the antioxidant activity turns out to have little to do with the breakdown of fat, it leads to many health benefits that will be discussed in greater detail in following sections.

    Green Tea and cancer

    Hundreds of studies have been done in recent years on the effect green tea has on both the prevention and treatment of cancer, as well as cardiovascular disease. Epidemiological studies tend to yield different results, with some finding no effect [27-28] and others finding that green tea drinkers have less incidence of certain types of cancer [29-30]. However, when a less specific variable such as total cancer deaths or lifespan is measured in a larger population it can be shown that green tea has a definite effect.

    One of the more comprehensive studies was conducted on a Japanese population over 13 years and the results were published this year in Ageing Research Reviews. It measured 90 lifestyle factors in a population of 8552 individuals over 40 years of age and determined the correlation of various factors with death from any cause. Consumption of over ten cups a day of green tea, when compared with less than three cups, was correlated with an increase in lifespan of 4.3 and 3.8 years in men and women respectively. Greater associations were seen in smokers (implying green tea had a protective effect) and those who died before age 80. Green tea was correlated with a 7.5 year longer lifespan in male smokers. Other large scale studies by this research group found that drinking green tea delayed cancer onset by 4.1 and 7.6 years in males and females respectively, cancer death by 3.9 and 5.9 years, and cardiovascular death by 1.9 and 1.4 years. Not surprisingly, these studies also found that smoking was correlated with earlier deaths from cancer and cardiovascular disease. The conclusion of this group was that green tea increases lifespan and decreases the risk of pre-mature death, particularly that caused by cancer [31].

    Many animal and in vitro studies have also been conducted on the effect green tea has on specific types of cancer. In mice, both oral and topical administration of green tea significantly decreases the risk of UV-induced skin cancer [32]. However, it is possible that this is due to decreased tissue fat [33], and caffeine is a necessary constituent in this case [17, 33], so more research is needed in this area. Green tea selectively destroys breast cancer cells [34, 35] and epidemiologic data implies that it aids in the prevention of early stage breast cancer [30]. A case control study also found that green tea decreases the risk of developing ovarian cancer [36], while other in vitro data has found that green tea inhibits the proliferation of cervical cancer [37], prostate cancer [38], leukemia [39], head and neck carcinoma cells [35], and pancreatic carcinoma cells [40]. In the case of lung cancer, green tea has a definite and significant effect in smokers and nonsmokers alike. In vitro data [41] and epidemiologic data in both smokers [42] and nonsmokers [29] support this. Finally, green tea may have the ability to prevent cancer in the gastrointestinal tract. An epidemiologic study in China found that green tea drinkers had less incidence of gastrointestinal cancer [43], although a different study in Japan found no statistically significant difference [28]. In vitro and in rats, polyphenols also protect against colon cancer [44, 45].

    As if this wasn't enough, green tea has also been tested against many carcinogens and almost always proves successful. Two toxins that humans are very commonly exposed to, tobacco and alcohol, are probably of most importance. An epidemiological study in China compared the incidence of various types of cancer among cigarette smokers and alcohol drinkers. In alcohol drinkers, those that regularly consumed green tea had 81%, 78%, and 39% decreased risk of gastric, liver and esophageal cancer, while smokers that drank green tea had 16%, 43%, and 31% decreased risk of these cancers [42]. In both mice and rats, green tea significantly protects against tumorgenesis induced by nitrosamines, which are found in tobacco as well as many food products [46, 47]. It also protects against the toxic effects of the environmental pollutant pentachlorphenol (PCP) and the chemotherapeutic drug cyclophosphamide (CP) in mice [48, 49] and the cancer induced by the carcinogens DMBA and DMH in other rodents [45, 50].

    As with the thermogenic effect, there are multiple mechanisms for the proctive effect green tea has against cancer, and many of them are tissue specific. One of the more important ones is the protection against oxidative stress, which may play a primary role in certain types of cancer [48, 49-52]. The antioxidant properties of green tea and their importance will be discussed in more detail later on. Of more importance is that when it comes to cancer cells, green tea is actually a pro-oxidant which results in the death of these cells [53]. This condition-dependent antioxidant/pro-oxidant duality is a common feature of many antioxidants.

    EGCG plays a fundamental role in cancer prevention as it inhibits many proteins and the activity of many protein kinases involved in tumor cell proliferation and survival. These include (but are by no means limited to) the large multi-catalytic protease and metaloproteionases involved in tumor survival and metastasis and the epidermal growth factor receptor (EGFR), vascular endothelial growth factor receptor (VEGF), platelet-derived growth factor receptor, mitogen-activated protein kinase, and IkB kinase [52]. The inhibition of EGFR and VEGF are of importance because they are known to play roles in angiogenesis [35, 28], which contributes to tumor formation in many cases [54]. The inhibition of metalloproteinases MMP-2 and MMP-9 are also known to play important roles in the anti-carcinogenic action of green tea [38, 41].

    Other benefits of Green Tea

    Many of the ingredients in green tea are potent antioxidants. In vitro, green tea and/or EGCG prevent the development of or directly quench a variety of reactive oxygen species (ROS) including superoxide [55], peroxynitrite [56, 57], and hydroxy radicals [57]. In preventing lipid peroxidation by hydrogen peroxide, it was superior to both lipoic acid and melatonin [58]. In humans, acute administration of green tea significantly improves plasma antioxidant capacity [59-61], with 450 mL (which would contain about 375 mg EGCG) causing an increase of 12.7% after two hours in one study [61]. In turn, this enhanced protection against oxidative stress offers a variety of health benefits.

    As discussed earlier, drinking green tea may delay death from cardiovascular disease by 1-2 years with a greater increase seen in men [31]. A study on 14,000 Japanese workers adjusted for dietary factors, age, BMI, alcohol consumption, tobacco use, coffee intake, and type of work found that green tea drinkers had significantly lower cholesterol levels [62] while a smaller study on 500 Japenese subjects found a statistically significant reduced risk of atherosclerosis in men, but not in women [63]. Although the present epidemiologic data does not support the idea that green tea increases HDL ("good") cholesterol in humans [62], it has been demonstrated that it does so in rats [64]. The data in this regard is still relatively preliminary so it is hard to draw too many conclusions, but the proposed mechanism of action for the delay in cardiovascular death is prevention of oxidative damage [57].

    Due to its ability to increase superoxide dismutase and glutathione levels, green tea also may aid in the prevention of insulin resistance and type II diabetes [65], which is often closely interrelated with other cardiovascular conditions. In normal rats, green tea significantly increases glucose tolerance, while in diabetic rats it significantly reduces serum glucose [65]. Another study also found that green tea improved kidney function in diabetic rats [66].

    Another area in which the activity of green tea is particularly important is in the brain. It goes without saying that green may protect against the development of brain tumors [67]. It also protects against oxidative damage in the brain [57] and improves brain recovery from ischemia/reperfusion injury in rats [68]. Green tea may also be useful in preventing Parkinson's disease through a fairly specific mechanism, and this has been an area of much study as of late [69].

    Green tea also exert a protective effect in the liver, acting in a synergistic fashion with vitamin E [70], as well as the digestive organs. It protects against or lessens liver damage caused by alcohol and carbon tetrachloride in rats [71, 72] and protects liver cells from a variety of toxins in vitro [73]. In the gastrointestinal tract, it reverses intestinal damage induced by fasting in rats [74] and inhibits the production of a toxin (produced by Helicobacter pyroli) associated with some gastric diseases [75]. Other preliminary studies indicate that green tea may be useful in the treatment of arthritis [76] and cataracts [77].

    Finally, recent cell culture studies have found that green tea may have strong antiviral activity. It has been tested sucessfully against influenza A and B and has been found to inhibit their growth [78], and it may also decrease the chance of HIV infection [79]. As of yet, it is unknown whether these observations are relevant in humans, in which the concentrations of the active ingredients wwould be much smaller.

    Possible side effects and precautions

    Green tea, even in large amounts, is associated with very few side effects. In mice, signs of toxicity were only observed when doses reached 2 g/kg daily of an 80% polyphenol abstract (this is about the equivalent of 8 cups per pound of body weight, per day – 1200 cups of tea in a 150 lb. individual) [80].

    It is possible that large amounts of polyphenols could impair mineral absorption, making extra mineral supplements a wise course of action. Of primary importance is a significant inhibition of nonheme iron absorption, which is relevant if most of your iron comes from sources other than meat [81, 82]. It is possible that there is also mild inhibition of calcium, manganese, and zinc [83, 84], although there is a study that disputes the finding of impaired zinc absorption [84]. The only place where the phenomenon of significant mineral blockage by green tea has even been of worry is in Tunisia, where there is high tea intake and non-meat products are the primary source of dietay iron [81]. However, it may be prudent to take a multivitamin supplement that provides adequate amounts of these minerals at a separate time or a low-dose slow release iron supplement if one decides to consume large amounts of polyphenols.

    A concern that is commonly brought up about green tea is the idea that it decreases levels of androgens such as testosterone and DHT. One study reported that after administered to rats, EGCG "significantly reduced food intake; body weight; blood levels of testosterone, estradiol, leptin, insulin, insulin-like growth factor I, LH, glucose, cholesterol, and triglyceride" [22]. However, a different study with green tea catechins in rats found decreased body weight but increased thyroid stimulating hormone (TSH), LH, and testosterone levels [85]. A final study in mice with green tea indicated drastic increases in both testosterone and DHT from green tea treatment, but levels of both were synergistically inhibited when it was adminstered along with soy phytochemicals [86]. In vitro, EGCG inhibits type I 5AR [2, 87], which is partially responsible for the conversion of testosterone to DHT (for this reason, it has been proposed in the topical treatment of acne and hair loss [2]), while in rats, green tea is an aromatase inhibitor, which (in theory) would be responsible for an increase in tesotsterone levels [85].

    So, how does one make sense of this mess of contadictory data? It should first be noted that 5AR inhibition has only been shown in vitro [87], while it has not been observed in live animals – in fact, drastic increases in DHT have been found - and it seems that other constituents of green tea counteract the 5AR inhibition [88]. The conclusions of the first study, which found drastic decreases in body weight, testosterone, and other hormones, are very misleading. A drastic reduction in levels of all of the substances mentioned can unfortunately be expected from weight loss in any situation (which is one of the reasons losing weight without losing muscle mass is so difficult). Indeed, when the authors of the same study restricted the food intake of the rats to cause a similar weight loss to that induced by EGCG, similar effects were seen, implying that the changes were not due to EGCG but due to drastic body weight loss [22]. The effect green tea has on levels of androgens and other hormones is complex and depends on various factors, but no detrimental effects have as of yet been seen in human populations.

    Green Tea pharmacokinetics

    This last section will examine the optimal dosage and dosing schedule for green tea. For cancer prevention, the present data indicate that relatively high amounts are required to achieve a significant effect – 10 cups or more per day [55, 88]. Luckily for those of us that do not have time to brew and drink ten cups of tea per day, there are supplements. (It has even been suggested that 10 cups a day in addition to supplements would be ideal for cancer prevention [89]). Ten cups is about 20 grams of tea leaves, which would contain about 2 grams of polyphenols, 1 gram of which would be EGCG. In comparison, 270 mg EGCG is all that is needed for significant fat loss [1], and this would still have positive health benefits. When we look at the pharmacokinetics of green tea, we can see that there may be ways to maximize the effect of a given amount. The most important thing to note is that green tea polyphenols undergo saturable presystemic elimination. This means that low doses only increase plasma EGCG levels marginally, but once the point of saturation is reached, the same amount of tea will have much greater effects.

    The amount required to achieve saturation in humans varies from study to study. Using tea leaves, one study found 3.0 grams to increase plasma EGCG levels by 2.7-3.4 times as much as 1.5 grams, while the difference between 3.0 grams and 4.5 grams was not statistically significant [90]. However, a study measuring antioxidant potential of green tea leaves found that 2.5, 5.0, and 7.5 increased plasma antioxidant potential by 2.1%, 6.2%, and 12.7% respectively at the 120 minute mark, indicating that 7.5 grams was over twice as effective as 5.0 grams [61]. 7.5 grams equates to about 375 mg of EGCG (note that this is only an approximation), and a pharmacokinetic study with green tea extract yielded a similar result. Amounts containing 225, 375, and 525 mg EGCG raised plamsa concentrations by 657, 4300, 4410 pmol/mL, respectively [91]. However, in a last study, despite the fact that 400 mg EGCG (from an extract) overcame saturation as opposed to 200 mg (the AUC, in this case a measure of total bioavailability, was 23.0 and 64.9 respectively), 600 mg and 800 mg amounts kept getting significantly more effective, with AUCs of 111.1 and 258.2 respectively [92]. The maximum plasma concentrations similarly increased. It is also noteworthy that this study found that it took 600, not 400 mg of pure EGCG (without other catechins) to overcome saturation, once again emphasizing that the other catechins operate synergistically with EGCG.

    With this data in mind, we could safetly say, for example, that taking an extract containing 400 mg EGCG once daily would be considerably more effective than 200 mg twice daily. Increasing this amount to 600 mg may or may not have a significant effect, depending on the study we look at. Either way, a minumum of 400 mg EGCG should be taken at a time to overcome saturation. An ideal dosing schedule would be 400 mg 2-3 times daily, while a more economical (but still very effective) one would be 400 mg once daily (preferably in the pre-workout period).


    LEAN FUEL XP


    References:

    1. Chantre P, Lairon D. Phytomedicine. Recent findings of green tea extract AR25 (Exolise) and its activity for the treatment of obesity. 2002 Jan;9(1):3-8 [pubmed]
    2. Liao S. The medicinal action of androgens and green tea epigallocatechin gallate. Hong Kong Med J. 2001 Dec;7(4):369-74 [pubmed]
    3. Morre DJ, Morre DM, Sun H, Cooper R, Chang J, Janle EM. Tea Catechin Synergies in Inhibition of Cancer Cell Proliferation and of a Cancer Specific Cell Surface Oxidase (ECTO-NOX). Pharmacol Toxicol. 2003 May;92(5):234-41 [pubmed]
    4. Kajiya K, Kumazawa S, Nakayama T. Effects of external factors on the interaction of tea catechins with lipid bilayers. Biosci Biotechnol Biochem. 2002 Nov;66(11):2330-5 [abstract] [pubmed]
    5. Ohe T, Marutani K, Nakase S. Catechins are not major components responsible for anti-genotoxic effects of tea extracts against nitroarenes. Mutat Res. 2001 Sep 20;496(1-2):75-81 [pubmed]
    6. Paquay JB, Haenen GR, Stender G, Wiseman SA, Tijburg LB, Bast A. Protection against nitric oxide toxicity by tea. J Agric Food Chem. 2000 Nov;48(11):5768-72 [pubmed]
    7. Higashi-Okai K, Yamazaki M, Nagamori H, Okai Y. J UOEH. Identification and antioxidant activity of several pigments from the residual green tea (Camellia sinensis) after hot water extraction. 2001 Dec 1;23(4):335-44 [abstract] [pubmed]
    8. Okai Y, Higashi-Okai K. Potent suppressing activity of the non-polyphenolic fraction of green tea (Camellia sinensis) against genotoxin-induced umu C gene expression in Salmonella typhimurium (TA 1535/pSK 1002)--association with pheophytins a and b. Cancer Lett. 1997 Nov 25;120(1):117-23 [pubmed]
    9. Higashi-Okai K, Okai Y. Potent suppressive activity of chlorophyll a and b from green tea (Camellia sinensis) against tumor promotion in mouse skin. J UOEH. 1998 Sep 1;20(3):181-8 [abstract] [pubmed]
    10. Yokogoshi H, Kobayashi M. Life Sci. Hypotensive effect of gamma-glutamylmethylamide in spontaneously hypertensive rats. 1998;62(12):1065-8 [pubmed]
    11. Kakuda T. Neuroprotective effects of the green tea components theanine and catechins. Biol Pharm Bull. 2002 Dec;25(12):1513-8 [pubmed]
    12. Sadzuka Y, Yamashita Y, Kishimoto S, Fukushima S, Takeuchi Y, Sonobe T. [Glutamate transporter mediated increase of antitumor activity by theanine, an amino acid in green tea]. Yakugaku Zasshi. 2002 Nov;122(11):995-9 [abstract] [pubmed]
    13. Sadzuka Y, Sugiyama T, Sonobe T. Improvement of idarubicin induced antitumor activity and bone marrow suppression by theanine, a component of tea. Cancer Lett. 2000 Oct 1;158(2):119-24 [pubmed]
    14. Yokogoshi H, Kobayashi M, Mochizuki M, Terashima T. Effect of theanine, r-glutamylethylamide, on brain monoamines and striatal dopamine release in conscious rats. Neurochem Res. 1998 May;23(5):667-73 [abstract] [pubmed]
    15. Mason R. 200 mg of Zen; L-theanine boosts alpha waves, promotes alert relaxation. Alternative & Complementary Therapies 2001 April; 7:91-95
    16. Kakuda T, Nozawa A, Unno T, Okamura N, Okai O. Inhibiting effects of theanine on caffeine stimulation evaluated by EEG in the rat. Biosci Biotechnol Biochem. 2000 Feb;64(2):287-93 [abstract] [pubmed]
    17. Lu YP, Lou YR, Li XH, Xie JG, Lin Y, Shih WJ, Conney AH. Stimulatory effect of topical application of caffeine on UVB-induced apoptosis in mouse skin. Oncol Res. 2002;13(2):61-70 [abstract] [pubmed]
    18. McArdle NJ, Clifford MN, Ioannides C. Consumption of tea modulates the urinary excretion of mutagens in rats treated with IQ. Role of caffeine. Mutat Res. 1999 May 17;441(2):191-203. [pubmed]
    19. Dulloo AG, Seydoux J, Girardier L, Chantre P, Vandermander J. Green tea and thermogenesis: interactions between catechin-polyphenols, caffeine and sympathetic activity. Int J Obes Relat Metab Disord. 2000 Feb;24(2):252-8 [abstract] [pubmed]
    20. Mori M, Hasegawa N. Superoxide dismutase activity enhanced by green tea inhibits lipid accumulation in 3T3-L1 cells. Phytother Res. 2003 May;17(5):566-7 [pubmed]
    21. Hasegawa N, Niimi N, Odani F. Vitamin C is one of the lipolytic substances in green tea. Phytother Res. 2002 Mar;16 Suppl 1:S91-2 [pubmed]
    22. Kao YH, Hiipakka RA, Liao S. Modulation of endocrine systems and food intake by green tea epigallocatechin gallate. Endocrinology. 2000 Mar;141(3):980-7 [pubmed]
    23. Sayama K, Lin S, Zheng G, Oguni I. Effects of green tea on growth, food utilization and lipid metabolism in mice. In Vivo. 2000 Jul-Aug;14(4):481-4 [abstract] [pubmed]
    24. Hasegawa N, Yamda N, Mori M. Powdered green tea has antilipogenic effect on Zucker rats fed a high-fat diet. Phytother Res. 2003 May;17(5):477-80 [pubmed]
    25. Dulloo AG, Duret C, Rohrer D, Girardier L, Mensi N, Fathi M, Chantre P, Vandermander J. Efficacy of a green tea extract rich in catechin polyphenols and caffeine in increasing 24-h energy expenditure and fat oxidation in humans. Am J Clin Nutr. 1999 Dec;70(6):1040-5 [pubmed]
    26. Yang M, Wang C, Chen H. Green, oolong and black tea extracts modulate lipid metabolism in hyperlipidemia rats fed high-sucrose diet. J Nutr Biochem. 2001 Jan;12(1):14-20 [pubmed]
    27. Nagano J, Kono S, Preston DL, Mabuchi K. A prospective study of green tea consumption and cancer incidence, Hiroshima and Nagasaki (Japan). Cancer Causes Control. 2001 Aug;12(6):501-8 [abstract] [pubmed]
    28. Hoshiyama Y, Kawaguchi T, Miura Y, Mizoue T, Tokui N, Yatsuya H, Sakata K, Kondo T, Kikuchi S, Toyoshima H, Hayakawa N, Tamakoshi A, Ohno Y, Yoshimura T; Japan Collaborative Cohort Study Group. A prospective study of stomach cancer death in relation to green tea consumption in Japan. Br J Cancer. 2002 Jul 29;87(3):309-13 [pubmed]
    29. Zhong L, Goldberg MS, Gao YT, Hanley JA, Parent ME, Jin F. A population-based case-control study of lung cancer and green tea consumption among women living in Shanghai, China. Epidemiology. 2001 Nov;12(6):695-700 [pubmed]
    30. Inoue M, Tajima K, Mizutani M, Iwata H, Iwase T, Miura S, Hirose K, Hamajima N, Tominaga S. Regular consumption of green tea and the risk of breast cancer recurrence: follow-up study from the Hospital-based Epidemiologic Research Program at Aichi Cancer Center (HERPACC), Japan. Cancer Lett. 2001 Jun 26;167(2):175-82 [pubmed]
    31. Nakachi K, Eguchi H, Imai K. Can teatime increase one's lifetime? Ageing Res Rev. 2003 Jan;2(1):1-10 [pubmed]
    32. Conney AH, Lu YP, Lou YR, Huang MT. Inhibitory effects of tea and caffeine on UV-induced carcinogenesis: relationship to enhanced apoptosis and decreased tissue fat. Eur J Cancer Prev. 2002 Aug;11 Suppl 2:S28-36 [pubmed]
    33. Lu YP, Lou YR, Lin Y, Shih WJ, Huang MT, Yang CS, Conney AH. Inhibitory effects of orally administered green tea, black tea, and caffeine on skin carcinogenesis in mice previously treated with ultraviolet B light (high-risk mice): relationship to decreased tissue fat. Cancer Res. 2001 Jul 1;61(13):5002-9 [pubmed]
    34. Vergote D, Cren-Olive C, Chopin V, Toillon RA, Rolando C, Hondermarck H, Le Bourhis X. (-)-Epigallocatechin (EGC) of green tea induces apoptosis of human breast cancer cells but not of their normal counterparts. Breast Cancer Res Treat. 2002 Dec;76(3):195-201 [abstract] [pubmed]
    35. Masuda M, Suzui M, Lim JT, Deguchi A, Soh JW, Weinstein IB. Epigallocatechin-3-gallate decreases VEGF production in head and neck and breast carcinoma cells by inhibiting EGFR-related pathways of signal transduction. J Exp Ther Oncol. 2002 Nov-Dec;2(6):350-9 [abstract] [pubmed]
    36. Zhang M, Binns CW, Lee AH. Tea consumption and ovarian cancer risk: a case-control study in China. Cancer Epidemiol Biomarkers Prev. 2002 Aug;11(8):713-8 [pubmed]
    37. Ahn WS, Huh SW, Bae SM, Lee IP, Lee JM, Namkoong SE, Kim CK, Sin JI. A major constituent of green tea, EGCG, inhibits the growth of a human cervical cancer cell line, CaSki cells, through apoptosis, G(1) arrest, and regulation of gene expression. DNA Cell Biol. 2003 Mar;22(3):217-24 [pubmed]
    38. Adhami VM, Ahmad N, Mukhtar H. Molecular targets for green tea in prostate cancer prevention. J Nutr. 2003 Jul;133(7):2417S-24S [pubmed]
    39. Lung HL, Ip WK, Wong CK, Mak NK, Chen ZY, Leung KN. Life Sci. Anti-proliferative and differentiation-inducing activities of the green tea catechin epigallocatechin-3-gallate (EGCG) on the human eosinophilic leukemia EoL-1 cell line. 2002 Dec 6;72(3):257-68 [pubmed]
    40. Takada M, Nakamura Y, Koizumi T, Toyama H, Kamigaki T, Suzuki Y, Takeyama Y, Kuroda Y. Suppression of human pancreatic carcinoma cell growth and invasion by epigallocatechin-3-gallate. Pancreas. 2002 Jul;25(1):45-8 [pubmed]
    41. Sartor L, Pezzato E, Garbisa S. (-)Epigallocatechin-3-gallate inhibits leukocyte elastase: potential of the phyto-factor in hindering inflammation, emphysema, and invasion. J Leukoc Biol. 2002 Jan;71(1):73-9 [pubmed]
    42. Mu LN, Zhou XF, Ding BG, Wang RH, Zhang ZF, Chen CW, Wei GR, Zhou XM, Jiang QW, Yu SZ. [A case-control study on drinking green tea and decreasing risk of cancers in the alimentary canal among cigarette smokers and alcohol drinkers]. Zhonghua Liu Xing Bing Xue Za Zhi. 2003 Mar;24(3):192-5 [abstract] [pubmed]
    43. Ye WM, Yi YN, Luo RX, Zhou TS, Lin RT, Chen GD. Diet and gastric cancer: a casecontrol study in Fujian Province, China. World J Gastroenterol. 1998 Dec;4(6):516-518 [pubmed]
    44. Chen C, Shen G, Hebbar V, Hu R, Owuor ED, Kong AN. Epigallocatechin-3-gallate-induced stress signals in HT-29 human colon adenocarcinoma cells. Carcinogenesis. 2003 Jun 19 [Epub ahead of print]. [pubmed]
    45. Jia X, Wang W, Cui W, Han C. [Effects of tea on aberrant crypt foci and colorectal tumors in rats] Wei Sheng Yan Jiu. 2000 Jan 30;29(1):54-6 [abstract] [pubmed]
    46. Zhu M, Gong Y, Yang Z. [Protective effect of tea on immune function in mice]. Zhonghua Yu Fang Yi Xue Za Zhi. 1998 Sep;32(5):270-4 [abstract] [pubmed]
    47. Sato D, Matsushima M. Preventive effects of urinary bladder tumors induced by N-butyl-N-(4-hydroxybutyl)-nitrosamine in rat by green tea leaves. Int J Urol. 2003 Mar;10(3):160-6 [pubmed]
    48. Edwin D, Geetha VR, Vishwanathan H, Usha Rani MV. An anticlastogenic in vivo micronucleus assay for tea. J Environ Biol. 2002 Oct;23(4):373-6 [abstract] [pubmed]
    49. Umemura T, Kai S, Hasegawa R, Kanki K, Kitamura Y, Nishikawa A, Hirose M. Prevention of dual promoting effects of pentachlorophenol, an environmental pollutant, on diethylnitrosamine-induced hepato- and cholangiocarcinogenesis in mice by green tea infusion. Carcinogenesis. 2003 Jun;24(6):1105-9. Epub 2003 Apr 24 [pubmed]
    50. Li N, Han C, Chen J. [Effects of tea on DMBA-induced oral carcinogenesis in hamsters] Wei Sheng Yan Jiu. 1999 Sep 30;28(5):289-92 [abstract] [pubmed]
    51. Hiroyasu M, Ozeki M, Miyagawa-Hayashino A, Fujiwara Y, Hiai H, Toyokuni S. Novel surrogate end-point biomarker to evaluate agents for use in the chemoprevention of reactive oxygen species-associated cancer. Redox Rep. 2002;7(5):335-8 [abstract] [pubmed]
    52. Kazi A, Smith DM, Daniel K, Zhong S, Gupta P, Bosley ME, Dou QP. Potential molecular targets of tea polyphenols in human tumor cells: significance in cancer prevention. In Vivo. 2002 Nov-Dec;16(6):397-403 [abstract] [pubmed]
    53. Malik A, Azam S, Hadi N, Hadi SM. DNA degradation by water extract of green tea in the presence of copper ions: implications for anticancer properties. Phytother Res. 2003 Apr;17(4):358-63 [pubmed]
    54. Pfeffer U, Ferrari N, Morini M, Benelli R, Noonan DM, Albini A. Antiangiogenic activity of chemopreventive drugs. Int J Biol Markers. 2003 Jan-Mar;18(1):70-4 [abstract] [pubmed]
    55. Nakagawa T, Yokozawa T. Direct scavenging of nitric oxide and superoxide by green tea. Food Chem Toxicol. 2002 Dec;40(12):1745-50 [pubmed]
    56. McConnell P, Reasor MJ, Dyke KV. Three model systems measure oxidation/nitration damage caused by peroxynitrite. J Biosci. 2003 Feb;28(1):71-6 [pubmed]
    57. Nagai K, Jiang MH, Hada J, Nagata T, Yajima Y, Yamamoto S, Nishizaki T. (-)-Epigallocatechin gallate protects against NO stress-induced neuronal damage after ischemia by acting as an anti-oxidant. Brain Res. 2002 Nov 29;956(2):319-22 [pubmed]
    58. Lee SR, Im KJ, Suh SI, Jung JG. Protective effect of green tea polyphenol (-)-epigallocatechin gallate and other antioxidants on lipid peroxidation in gerbil brain homogenates. Phytother Res. 2003 Mar;17(3):206-9 [pubmed]
    59. Serafini M, Laranjinha JA, Almeida LM, Maiani G. Inhibition of human LDL lipid peroxidation by phenol-rich beverages and their impact on plasma total antioxidant capacity in humans. J Nutr Biochem. 2000 Nov;11(11-12):585-590 [pubmed]
    60. Leenen R, Roodenburg AJ, Tijburg LB, Wiseman SA. A single dose of tea with or without milk increases plasma antioxidant activity in humans. Eur J Clin Nutr. 2000 Jan;54(1):87-92 [abstract] [pubmed]
    61. Sung H, Nah J, Chun S, Park H, Yang SE, Min WK. In vivo antioxidant effect of green tea. Eur J Clin Nutr. 2000 Jul;54(7):527-9 [abstract] [pubmed]
    62. Tokunaga S, White IR, Frost C, Tanaka K, Kono S, Tokudome S, Akamatsu T, Moriyama T, Zakouji H. Green tea consumption and serum lipids and lipoproteins in a population of healthy workers in Japan. Ann Epidemiol. 2002 Apr;12(3):157-65 [pubmed]
    63. Sasazuki S, Kodama H, Yoshimasu K, Liu Y, Washio M, Tanaka K, Tokunaga S, Kono S, Arai H, Doi Y, Kawano T, Nakagaki O, Takada K, Koyanagi S, Hiyamuta K, Nii T, Shirai K, Ideishi M, Arakawa K, Mohri M, Takeshita A. Relation between green tea consumption and the severity of coronary atherosclerosis among Japanese men and women. Ann Epidemiol. 2000 Aug;10(6):401-8 [pubmed]
    64. Yokozawa T, Nakagawa T, Kitani K. Antioxidative activity of green tea polyphenol in cholesterol-fed rats. J Agric Food Chem. 2002 Jun 5;50(12):3549-52 [pubmed]
    65. M C S, K S, Kuttan R. Anti-diabetic activity of green tea polyphenols and their role in reducing oxidative stress in experimental diabetes. J Ethnopharmacol. 2002 Nov;83(1-2):109-16 [pubmed]
    66. Rhee SJ, Kim MJ, Kwag OG. Effects of green tea catechin on prostaglandin synthesis of renal glomerular and renal dysfunction in streptozotocin-induced diabetic rats. Asia Pac J Clin Nutr. 2002;11(3):232-6 [pubmed]
    67. Yokoyama S, Hirano H, Wakimaru N, Sarker KP, Kuratsu J. Inhibitory effect of epigallocatechin-gallate on brain tumor cell lines in vitro. Neuro-oncol. 2001 Jan;3(1):22-8 [pubmed]
    68. Hong JT, Ryu SR, Kim HJ, Lee JK, Lee SH, Kim DB, Yun YP, Ryu JH, Lee BM, Kim PY. Neuroprotective effect of green tea extract in experimental ischemia-reperfusion brain injury. Brain Res Bull. 2000 Dec;53(6):743-9 [pubmed]
    69. Pan T, Fei J, Zhou X, Jankovic J, Le W. Effects of green tea polyphenols on dopamine uptake and on MPP+ -induced dopamine neuron injury. Life Sci. 2003 Jan 17;72(9):1073-83 [pubmed]
    70. Cai YJ, Ma LP, Hou LF, Zhou B, Yang L, Liu ZL. Antioxidant effects of green tea polyphenols on free radical initiated peroxidation of rat liver microsomes. Chem Phys Lipids. 2002 Dec;120(1-2):109-17 [pubmed]
    71. Skrzydlewska E, Ostrowska J, Stankiewicz A, Farbiszewski R. Green tea as a potent antioxidant in alcohol intoxication. Addict Biol. 2002 Jul;7(3):307-14 [abstract] [pubmed]
    72. Xiao J, Lu R, Shen X, Wu M. [Green tea extracts protected against carbon tetrachloride-induced chronic liver damage and cirrhosis] Zhonghua Yu Fang Yi Xue Za Zhi. 2002 Jul;36(4):243-6 [abstract] [pubmed]
    73. Kagaya N, Tagawa Y, Nagashima H, Saijo R, Kawase M, Yagi K. Suppression of cytotoxin-induced cell death in isolated hepatocytes by tea catechins. Eur J Pharmacol. 2002 Aug 30;450(3):231-6 [pubmed]
    74. Asfar S, Abdeen S, Dashti H, Khoursheed M, Al-Sayer H, Mathew T, Al-Bader A. Effect of green tea in the prevention and reversal of fasting-induced intestinal mucosal damage. Nutrition. 2003 Jun;19(6):536-40 [pubmed]
    75. Tombola F, Campello S, De Luca L, Ruggiero P, Del Giudice G, Papini E, Zoratti M. Plant polyphenols inhibit VacA, a toxin secreted by the gastric pathogen Helicobacter pylori. FEBS Lett. 2003 May 22;543(1-3):184-9 [pubmed]
    76. Ahmed S, Rahman A, Hasnain A, Lalonde M, Goldberg VM, Haqqi TM. Green tea polyphenol epigallocatechin-3-gallate inhibits the IL-1 beta-induced activity and expression of cyclooxygenase-2 and nitric oxide synthase-2 in human chon
    drocytes. Free Radic Biol Med. 2002 Oct 15;33(8):1097-105 [pubmed]
    77. Gupta SK, Halder N, Srivastava S, Trivedi D, Joshi S, Varma SD. Green tea (Camellia sinensis) protects against selenite-induced oxidative stress in experimental cataractogenesis. Ophthalmic Res. 2002 Jul-Aug;34(4):258-63 [pubmed]
    78. Imanishi N, Tuji Y, Katada Y, Maruhashi M, Konosu S, Mantani N, Terasawa K, Ochiai H. Additional inhibitory effect of tea extract on the growth of influenza A and B viruses in MDCK cells. Microbiol Immunol. 2002;46(7):491-4 [abstract] [pubmed]
    79. Weber JM, Ruzindana-Umunyana A, Imbeault L, Sircar S. Inhibition of adenovirus infection and adenain by green tea catechins. Antiviral Res. 2003 Apr;58(2):167-73 [pubmed]
    80. Chang PY, Mirsalis J, Riccio ES, Bakke JP, Lee PS, Shimon J, Phillips S, Fairchild D, Hara Y, Crowell JA. Genotoxicity and toxicity of the potential cancer-preventive agent polyphenon E. Environ Mol Mutagen. 2003;41(1):43-54 [pubmed]
    81. Hamdaoui M, Hedhili A, Doghri T, Tritar B. Effect of tea on iron absorption from the typical Tunisian meal 'couscous' fed to healthy rats. Ann Nutr Metab. 1994;38(4):226-31 [abstract] [pubmed]
    82. Samman S, Sandstrom B, Toft MB, Bukhave K, Jensen M, Sorensen SS, Hansen M. Green tea or rosemary extract added to foods reduces nonheme-iron absorption. Am J Clin Nutr. 2001 Mar;73(3):607-12 [pubmed]
    83. Zeyuan D, Bingying T, Xiaolin L, Jinming H, Yifeng C. Effect of green tea and black tea on the metabolisms of mineral elements in old rats. Biol Trace Elem Res. 1998 Oct;65(1):75-86 [abstract] [pubmed]
    84. Record IR, McInerney JK, Dreosti IE. Black tea, green tea, and tea polyphenols. Effects on trace element status in weanling rats. Biol Trace Elem Res. 1996 Summer;53(1-3):27-43 [abstract] [pubmed]
    85. Satoh K, Sakamoto Y, Ogata A, Nagai F, Mikuriya H, Numazawa M, Yamada K, Aoki N. Inhibition of aromatase activity by green tea extract catechins and their endocrinological effects of oral administration in rats. Food Chem Toxicol. 2002 Jul;40(7):925-33 [pubmed]
    86. Zhou JR, Yu L, Zhong Y, Blackburn GL. Soy phytochemicals and tea bioactive components synergistically inhibit androgen-sensitive human prostate tumors in mice. J Nutr. 2003 Feb;133(2):516-21 [pubmed]
    87. Hiipakka RA, Zhang HZ, Dai W, Dai Q, Liao S. Structure-activity relationships for inhibition of human 5alpha-reductases by polyphenols. Biochem Pharmacol. 2002 Mar 15;63(6):1165-76 [pubmed]
    88. Kohlmeier L, Weterings KG, Steck S, Kok FJ. Tea and cancer prevention: an evaluation of the epidemiologic literature. Nutr Cancer. 1997;27(1):1-13 [abstract] [pubmed]
    89. Fujiki H, Suganuma M, Imai K, Nakachi K. Green tea: cancer preventive beverage and/or drug. Cancer Lett. 2002 Dec 15;188(1-2):9-13 [pubmed]
    90. Yang CS, Chen L, Lee MJ, Balentine D, Kuo MC, Schantz SP. Blood and urine levels of tea catechins after ingestion of different amounts of green tea by human volunteers. Cancer Epidemiol Biomarkers Prev. 1998 Apr;7(4):351-4 [abstract] [pubmed]
    91. Nakagawa K, Okuda S, Miyazawa T. Dose-dependent incorporation of tea catechins, (-)-epigallocatechin-3-gallate and (-)-epigallocatechin, into human plasma. Biosci Biotechnol Biochem. 1997 Dec;61(12):1981-5 [abstract] [pubmed]
    92. Chow HH, Cai Y, Alberts DS, Hakim I, Dorr R, Shahi F, Crowell JA, Yang CS, Hara Y. Phase I pharmacokinetic study of tea polyphenols following single-dose administration of epigallocatechin gallate and polyphenon E. Cancer Epidemiol Biomarkers Prev. 2001 Jan;10(1):53-8 [pubmed]





    IronMagLabs 15% Discount Code: Robert15



    IronMag Research 15% Discount Code: Robert15



  5. #5
    Is Doin It 4 Da Shorteez
    LAM's Avatar


    Join Date
    May 2002
    Gender
    Male
    Location
    Las Vegas & Florida & St. Croix
    Posts
    16,354
    Rep Points
    962148245

    Quote Originally Posted by Rocky_B
    Right now, for the first time in my life I can see an outline of my abs, but they aren't as defined as I'd like. I'm doing weighted and non-weighted ab exercises frequently throughout the week while at the gym and at home, and my diet is in check, and I run 3.5 kilometres about 2-3 times a week, in addition to training with weights in the gym 5 days a week. I take Whey, just started creatine for the first time less than a week ago (not loading), multis and fish oils, but haven't tried green tea extract. I'm lean but will green tea extract help in defining my abs further?
    you would do better runnning for a shorter period of time at a higher level of intensity. you will burn more fat after running a fast mile several times a week then after a moderately paced several miles. running shorter distance is also better in terms of the effects that it has on your endocrine system. distance running will cause higher levels of cortisol for several hours after the training session
    William F. Buckley describes a conservative as, "someone who stands athwart history, yelling Stop." - and then proceeds to drag civilization back to times best left in history's dungheap.

  6. #6
    Registered User
    Rocky_B's Avatar


    Join Date
    Jul 2004
    Location
    Canada
    Posts
    446
    Rep Points
    10

    Thanks
    IMA BEAST

  7. #7
    Registered User
    ricky_rocket's Avatar


    Join Date
    Feb 2005
    Posts
    125
    Rep Points
    10






    Sounds like you are in pretty good shape and just want to lose a little bit more fat. Try running in the morning on an empty stomach. Or take green tea caps in the morning and run before you eat. I'll but you'll burn off the unwanted fat arround the abs.

    (also, the green tea will have minimal effect. It's the running that burns the fat).

Similar Threads

  1. What's the Best Green Tea Extract?
    By SJ69 in forum Supplements
    Replies: 10
    Last Post: 07-21-2008, 02:28 PM
  2. Green tea extract, caffeine, and carnitine
    By Perk1975 in forum Supplements
    Replies: 7
    Last Post: 10-29-2007, 10:11 AM
  3. Best time to take green tea extract?
    By ddawg in forum Supplements
    Replies: 14
    Last Post: 02-19-2006, 09:22 AM
  4. Green tea extract
    By Nate K in forum Supplements
    Replies: 1
    Last Post: 11-02-2005, 11:18 AM
  5. Green Tea Extract
    By Fire_Woman in forum Diet & Nutrition
    Replies: 2
    Last Post: 03-03-2005, 03:35 PM

Posting Permissions

  • You may not post new threads
  • You may not post replies
  • You may not post attachments
  • You may not edit your posts
  •  
DISABLED END -->