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Fina ????

Hefty

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If i do no get fina cough what does that mean is it fake? and how long into cycle should i expect to get it??
 
I never had fina cough, fina breath yes, if I didn't do 100mg in a shot though I didn't really notice it.
 
do a shot of 200mg... heh... u will defintly cough.. well maybe not.. but i sure as hell did
 
I didn't get the cough on fina for my first two 8 wk fina cycles. Then one day I got it in my third cycle and I got it EVERY time after that. The only way I could defeat it was to mix the fina with another AAS.
 
Anyone know WHY fina would cause you to cough? It's not like your smoking the shyt? Fluid in the lungs maybe??
 
We can only guess nobody knows. Since its not intended for human use in the first place, scientists aren't exactly dying to find out for us.
 
Most recent beliefs are that PGF2a is toxic to the lungs and causes the cough. Pheedno made a couple of posts no one understands (at least me) relating to the topic on a few boards.
 
Tren- Fat burning and "fina cough" both from prostaglandin metabolization
Posted on AR & IBB as well-


It's been widely disussed of Trens fat burning properties through rises in IGF and Prostaglandins. While IGF is a fairly well known substance in the bodybuilding world today, prostaglandins are fairly unknown in terms of formation and roles in the body.
So below, a brief dicription of prostoglandins and their role in fat burning, "fina cough", and why a person going through Tren administration can experience it's fat burning effects without the dreaded "Cough"

The term prostaglandin comes from the word-Prostate. The first prostoglandins were first dicovered in semen about the mid 1930's and it was thought that prostaglandins were made from the prostate. Since this time, it has been dicovered that most prostaglandins are not even constructed in the prostate.

Prostaglandins are made by two different pathways(Cyclooxygenase and Lipoxygenase), and considering prostaglandins are a group of about 20 lipid cells, they have contrary function; responsible for stimulating as well as alleviating inflammation(Inflammation stimulation is the rapid metabolism of them expelled through the bronchials), regulate blood flow to particular organs, control ion transport across membranes, modulate synaptic transmission, induce sleep, mediate lipid release, and regulate metabolism is various tissue.

Prostaglandins are synthesized from arachidonate(Lipoxygenase which catalyze the dioxygenation of polyunsaturated fatty acids) in the cell membrane by the action of phospholipase A2. Cyclooxygenase and lipoxygenase pathways, compete with one another to form prostaglandins(as well as thromboxane or leukotriene-leukotriene being a bronchial stimulator),
In the cyclooxygenase pathway, the prostaglandins D, E and F plus thromboxane and prostacyclin are made. Thromboxanes are made in platelets and cause constriction of vascular smooth muscle and platelet aggregation
Leukotrienes are made in leukocytes and macrophages via the lipoxygenase pathway. They are potent constrictors of the bronchial airways. They are also important in inflammation and hypersensitivity reactions as they increase vascular permeability.

Being that prostaglandins from either pathway, are still fatty acids of a group, they mediate lipid release and controll tissue metabolization, so fat burning is a luxerry of either pathway of formation. It's the pathway from which they are constructed that dictates "fina cough". As prostaglandins made from the Cyclooxygenase pathway dictate muscle constriction and platlet aggregation, and the Lipoxygenase pathway dictates bronchial constriction(the main form of expulsion)


Refs:
Cackatoo Press
Columbia Encyclopedia 6th Edition
Science Daily Magazine
 
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