Least/Most Favorite Days + Strength Question

[Nate K]

I talked to my bio professor about that and her opinion was that hyerplasia of muscle cells actually occurs. I agree, the issue would be proving something like that on living human tissue without violating some kind of law.

I would agree....but shouldn't "they" be able to figure this out.....damn research regulations. I'm wondering if maybe muscle cells are just like neurons (brain cells) . I think you cannot increase the number of "neural pathways" but the body can develp stronger, more various or efficient connections and synapses along the way.

 

[CowPimp]

I believe that both the speed (the arc) and the strength are unchangeable (an assumption). I was thinking that more power stokes would be generated more often (faster recruitment). Additionally, since the power strokes are staggared (for smoothness), that perhaps the synchronization could be improved as well.

This is definitely the case. It is well established that rate coding, what you referring to as faster recruitment, does improve. As well, better motor unit synchronization does occur. My understanding is that rate coding contributes much more to increases in strength than MU synchronization, but it also appears to me that studies confirming this are fairly limited.

So, I guess that statement of not knowing about synchronization in my last post was a brain fart. I have definitely read about it from multiple sources.

I talked to my bio professor about that and her opinion was that hyerplasia of muscle cells actually occurs. I agree, the issue would be proving something like that on living human tissue without violating some kind of law.

I know there is evidence of hyperplasia occuring in animal studies. We all know that doesn't directly correlate to humans, but it should be taken with some type of merit I think. As well, things may also become different if you start factoring in anabolics into the equation.

 

[Doublebase]

I like bench press. Always been a comfortable press for me. I also enjoy standing overhead press. Sometimes I dread leg day.

 

[Duncans Donuts]

I know there is evidence of hyperplasia occuring in animal studies. We all know that doesn't directly correlate to humans, but it should be taken with some type of merit I think. As well, things may also become different if you start factoring in anabolics into the equation.

Well, as far as muscle physiology is generally concerned, there should be no reason (in my understanding) why humans are operating under different rules regarding musculature relative to other animals.

If you subscribe to design theory or evolutionary theory, the notion that if one animal can incur hyperplasia of muscle cells, the rest likely can too, fits.

Also, there are chemical reactions that occur when generating new tissue in a body (net protein turnover, Ck levels, blah blah), I'm pretty sure one can find the byproducts of manufacturing new muscle cells and use such data in a beneficial way to decide if it occurs in humans...who knows I'm not on the forefront of understanding this stuff.

 

[Dale Mabry]

I talked to my bio professor about that and her opinion was that hyerplasia of muscle cells actually occurs. I agree, the issue would be proving something like that on living human tissue without violating some kind of law.

I think a majority of people have this opinion.

Also, wrt muscle memory, I seem to remember reading something on the changes with synapse area in the essentials book. I know strength training causes a change in the shape of the NMJ leading to a greater surface area for ACh interaction which is partially responsible for the increase in ACh receptor activity. Not positive, but endurance training leads to a more symmetrical NMJ while resistance training leads to a wildly asymmetrical NMJ that is wavy and wide. I don't know if it changes back to normal after detraining, but I imagine it would take a significant amount of time to go back.

My guess is that it has more to do with having the movement pattern stored and being able to retrieve it moreso than a structural issue.

 

[Nate K]

I think a majority of people have this opinion.

Also, wrt muscle memory, I seem to remember reading something on the changes with synapse area in the essentials book. I know strength training causes a change in the shape of the NMJ leading to a greater surface area for ACh interaction which is partially responsible for the increase in ACh receptor activity. Not positive, but endurance training leads to a more symmetrical NMJ while resistance training leads to a wildly asymmetrical NMJ that is wavy and wide. I don't know if it changes back to normal after detraining, but I imagine it would take a significant amount of time to go back.

My guess is that it has more to do with having the movement pattern stored and being able to retrieve it moreso than a structural issue.

Makes sense..........nice info. man, thanks

 

[Pedro TT]

I love working out legs/ shoulders/ arms as its great feeling like total jello whenever you get out of the gym. I absolutely HATE working out abs. Me and and the midsection have never really got along very well

 

[CowPimp]

Well, as far as muscle physiology is generally concerned, there should be no reason (in my understanding) why humans are operating under different rules regarding musculature relative to other animals.

If you subscribe to design theory or evolutionary theory, the notion that if one animal can incur hyperplasia of muscle cells, the rest likely can too, fits.

Also, there are chemical reactions that occur when generating new tissue in a body (net protein turnover, Ck levels, blah blah), I'm pretty sure one can find the byproducts of manufacturing new muscle cells and use such data in a beneficial way to decide if it occurs in humans...who knows I'm not on the forefront of understanding this stuff.

I agree. I generally like to see hard evidence before I fully subscribe to something, but it certainly shouldn't be ruled out.

I think a majority of people have this opinion.

Also, wrt muscle memory, I seem to remember reading something on the changes with synapse area in the essentials book. I know strength training causes a change in the shape of the NMJ leading to a greater surface area for ACh interaction which is partially responsible for the increase in ACh receptor activity. Not positive, but endurance training leads to a more symmetrical NMJ while resistance training leads to a wildly asymmetrical NMJ that is wavy and wide. I don't know if it changes back to normal after detraining, but I imagine it would take a significant amount of time to go back.

My guess is that it has more to do with having the movement pattern stored and being able to retrieve it moreso than a structural issue.

Yeah, although I didn't know all those details, that was kind of my thought: the nervous system potentiating muscular hypertrophy.

 

[Dale Mabry]

Well now you know, and knowing is half the battle.

 

[DOMS]

Yes...yes it is.

 

[topolo]

Well now you know, and knowing is half the battle.

What's the other half?