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Been on for 6months, plateaued for a month switched up my cycle gaining like crazy

The shape I was in my avi was all due to mike arnold I was 270 pound fat fuck although freakishly strong he chiseled away at me to unveil my shape and I've progressed from that condition with all credit being giving to mike arnold.

Mike is a hairy legend around here. Writes a great essay as well!
 
also just for reference sake;

HCG myth debunked:


There are 2 ways that could potentially desensitize Leydig Cells:

1. Prolonged LH deprivation: When you inject steroids, your LH production is halted at the pituitary, remember? So if you continue in a suppressed state for weeks upon weeks, your Leydig Cells could potentially become unresponsive, or desensitized. It is possible to reverse desensitization of the cells, but that has been proven to be quite a difficult task. So when you use hCG on cycle, the mimicked LH analog will maintain stimulation of Leydig cells so that you don't run the risk of rendering them useless. This level of maintenance will ensure a much healthier and speedy recovery and one of the most important reasons to use hCG on cycle.

2. Over stimulation/supplying of Leydig cells: There is no reason to use more than 500 IU of hCG at one time. And certainly not a good idea to run even that dose on a daily basis. You do not have an unlimited-ever-flowing-supply of Leydig cells. There is only so much stimulation hCG can do. What happens when you dose hCG really high, is that you're increasing intra-testicular estrogen. So you're thinking that you could use an aromatase inhibitor in that case, right? Nope. AI's are not effective treatment for intra-testicular e2. Furthermore; high doses is a surefire way to desensitize Leydig Cells. So we have a double whammy here. And this is just another reason to use hCG on cycle, and not "blast" hCG post cycle leading up to and/or during PCT.

For the sake of preventing another debate, Rich Piana is clueless.
biggiesmallz said:
Now, I understand the proper usage of HCG on-cycle... generally advised at 250iu bi-weekly, sometimes at 500iu bi-weekly, but from the source I came across they referenced a study that basically said there's marginal benefit from HCG when pinning 250 vs 500 bi-weekly, so with that understanding I don't see the need to pin more than 250. That said, is there some limited duration to which HCG should be used on-cycle?
I also heard conflicting information on long-term HCG use can possibly desensitize lydig cells to natural LH response. Any truth to that?





There's only so many cells to stimulate, and the doses of 1500 weekly max, spread over 3 or more doses is sufficient enough. If long term therapy was dangerous at those doses, it would mean that our very own production would desensitize cells, doesnt make sense, does it? 250 IU is not necessarily the magic number. Your goal should be to use the least amount of hCG that works for you. Recently, discussing my concerns with the lead urologist in (some magical place out there somewhere), we came to conclude that for me, as a TRT patient, my usual dose of 250 twice weekly is excessive. So we are planning on reducing the dose to 100 IU, 3 times weekly. Note that this urologist is not my doctor, but a friend and partner in a clinical trial.

Blasting hCG is unhealthy, and the increase in intratesticular E2, which cannot be managed with the commonly readily available aromatase inhibitors, is damaging.
 
I was advised less then 300 isn't worth fuckin with. If you never ran tren at 500 or better for at least 8 weeks you don't know what your missin. I get night sweats once in a while other then that I'm good but I must say I'm very gunk prone so I keep that in check but I get no sides from juice. I've never broken out not once ever.

What is gunk prone?

"May the force be with you all"
 
also just for reference sake;

HCG myth debunked:


There are 2 ways that could potentially desensitize Leydig Cells:

1. Prolonged LH deprivation: When you inject steroids, your LH production is halted at the pituitary, remember? So if you continue in a suppressed state for weeks upon weeks, your Leydig Cells could potentially become unresponsive, or desensitized. It is possible to reverse desensitization of the cells, but that has been proven to be quite a difficult task. So when you use hCG on cycle, the mimicked LH analog will maintain stimulation of Leydig cells so that you don't run the risk of rendering them useless. This level of maintenance will ensure a much healthier and speedy recovery and one of the most important reasons to use hCG on cycle.

2. Over stimulation/supplying of Leydig cells: There is no reason to use more than 500 IU of hCG at one time. And certainly not a good idea to run even that dose on a daily basis. You do not have an unlimited-ever-flowing-supply of Leydig cells. There is only so much stimulation hCG can do. What happens when you dose hCG really high, is that you're increasing intra-testicular estrogen. So you're thinking that you could use an aromatase inhibitor in that case, right? Nope. AI's are not effective treatment for intra-testicular e2. Furthermore; high doses is a surefire way to desensitize Leydig Cells. So we have a double whammy here. And this is just another reason to use hCG on cycle, and not "blast" hCG post cycle leading up to and/or during PCT.

For the sake of preventing another debate, Rich Piana is clueless.

There's only so many cells to stimulate, and the doses of 1500 weekly max, spread over 3 or more doses is sufficient enough. If long term therapy was dangerous at those doses, it would mean that our very own production would desensitize cells, doesnt make sense, does it? 250 IU is not necessarily the magic number. Your goal should be to use the least amount of hCG that works for you. Recently, discussing my concerns with the lead urologist in (some magical place out there somewhere), we came to conclude that for me, as a TRT patient, my usual dose of 250 twice weekly is excessive. So we are planning on reducing the dose to 100 IU, 3 times weekly. Note that this urologist is not my doctor, but a friend and partner in a clinical trial.

Blasting hCG is unhealthy, and the increase in intratesticular E2, which cannot be managed with the commonly readily available aromatase inhibitors, is damaging.

I like it bro.. good info here

Sent from my SAMSUNG-SGH-I337 using Tapatalk
 
also just for reference sake;

HCG myth debunked:


There are 2 ways that could potentially desensitize Leydig Cells:

1. Prolonged LH deprivation: When you inject steroids, your LH production is halted at the pituitary, remember? So if you continue in a suppressed state for weeks upon weeks, your Leydig Cells could potentially become unresponsive, or desensitized. It is possible to reverse desensitization of the cells, but that has been proven to be quite a difficult task. So when you use hCG on cycle, the mimicked LH analog will maintain stimulation of Leydig cells so that you don't run the risk of rendering them useless. This level of maintenance will ensure a much healthier and speedy recovery and one of the most important reasons to use hCG on cycle.

2. Over stimulation/supplying of Leydig cells: There is no reason to use more than 500 IU of hCG at one time. And certainly not a good idea to run even that dose on a daily basis. You do not have an unlimited-ever-flowing-supply of Leydig cells. There is only so much stimulation hCG can do. What happens when you dose hCG really high, is that you're increasing intra-testicular estrogen. So you're thinking that you could use an aromatase inhibitor in that case, right? Nope. AI's are not effective treatment for intra-testicular e2. Furthermore; high doses is a surefire way to desensitize Leydig Cells. So we have a double whammy here. And this is just another reason to use hCG on cycle, and not "blast" hCG post cycle leading up to and/or during PCT.

For the sake of preventing another debate, Rich Piana is clueless.

There's only so many cells to stimulate, and the doses of 1500 weekly max, spread over 3 or more doses is sufficient enough. If long term therapy was dangerous at those doses, it would mean that our very own production would desensitize cells, doesnt make sense, does it? 250 IU is not necessarily the magic number. Your goal should be to use the least amount of hCG that works for you. Recently, discussing my concerns with the lead urologist in (some magical place out there somewhere), we came to conclude that for me, as a TRT patient, my usual dose of 250 twice weekly is excessive. So we are planning on reducing the dose to 100 IU, 3 times weekly. Note that this urologist is not my doctor, but a friend and partner in a clinical trial.

Blasting hCG is unhealthy, and the increase in intratesticular E2, which cannot be managed with the commonly readily available aromatase inhibitors, is damaging.

So in your opinion how would this translate into fertility(already desensitized leydig cells)? Its common for doctors to prescribe 1500 IU's three times a week and higher?
 
So in your opinion how would this translate into fertility(already desensitized leydig cells)? Its common for doctors to prescribe 1500 IU's three times a week and higher?

Doesn't E2 become a factor at such high doses?

Sent from my SAMSUNG-SGH-I337 using Tapatalk
 
Last edited:
So how much hcg should I take and it's not dose dependent on how much gear I'm on?
 
I usually dose it the last two weeks of cycle before PCT at 1000ius week.

Sent from my SAMSUNG-SGH-I337 using Tapatalk
 
Btw even tho I was using is it month on month off I wouldn't consider it a blast due to the face I was pinning 500 bi weekly..
 
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