http://www.orbitnutrition.com/cart/black-lion-research-prolactrone.html
Prolactrone up for pre orders.
Prolactrone up for pre orders.
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Blr prolactrone
- Thread starter OrbitNutrition
- Start date
brundel
Registered
Yes sir. By next week we will be full steam again with no lags in product.
I didnt anticipate things selling so fast.
At any rate:
Prolactrone has been improved vastly and the price has been dropped by over 10$
Its 166mg of a 99% ldopa and
333mg Green tea standardized for 50% ECGC
This stuff is pretty awesome.
It can replace Caber for tren and deca cycles.
It also will increase HGH levels
elevate mood
improve sleep
burn some fat.
For now there are limited pre orders.
Everyone should have full stock by the end of the week.
I didnt anticipate things selling so fast.
At any rate:
Prolactrone has been improved vastly and the price has been dropped by over 10$
Its 166mg of a 99% ldopa and
333mg Green tea standardized for 50% ECGC
This stuff is pretty awesome.
It can replace Caber for tren and deca cycles.
It also will increase HGH levels
elevate mood
improve sleep
burn some fat.
For now there are limited pre orders.
Everyone should have full stock by the end of the week.
:d:d
packers6211
Registered
Orbit pimp slapping hand is on fire!! Glad to see this coming back!!
brundel
Registered
Yah the goal with this prod is to have a legit alternative to research chem Caber.
The shits so hit and miss and 2x the price.
Prolactrone is super strong.
Most companies skimp and use 15-25% ldopa extracts. We use 99%
The shits potent and we have a rather large dose.
DO NOT EXCEED RECOMMENDED DOSE.
There is also drug interaction info on our site so make sure to read before using the product.
The ECGC prevents the ldopa from becoming dopamine outside the brain.
EGCG is a (decarboxylase inhibitor) Its also a COMT inhibitor (catechol-O-methyl transferase inhibitor)
Once inside dopamine goes up and prolactin goes down.
The shits so hit and miss and 2x the price.
Prolactrone is super strong.
Most companies skimp and use 15-25% ldopa extracts. We use 99%
The shits potent and we have a rather large dose.
DO NOT EXCEED RECOMMENDED DOSE.
There is also drug interaction info on our site so make sure to read before using the product.
The ECGC prevents the ldopa from becoming dopamine outside the brain.
EGCG is a (decarboxylase inhibitor) Its also a COMT inhibitor (catechol-O-methyl transferase inhibitor)
Once inside dopamine goes up and prolactin goes down.
brundel
Registered
I took a cap yesterday at work and I felt pretty good by the end of the day.
These are real "feel good" caps.
These are real "feel good" caps.
good info brundel, i gotta take a look at this!
brundel
Registered
Pre orders are almost sold out....day 2.
Looks like Im gonna be working alot......
This product is gonna be a staple soon.
Looks like Im gonna be working alot......
This product is gonna be a staple soon.
BFHammer
Registered
How would the dopamine part affect someone with ADHD?
brundel
Registered
Unsure. Maybe look into ldopa and adhd.
I really hope this helps with sleep, I ordered 2 bottles.
brundel
Registered
Ldopa is known to improve sleep.
It should help.
It should help.
brundel
Registered
After a quick browse from my phone it looks like ldopa is a treatment for adhd.
Katherinejjm
Banned Member
all hail orbit and brundel
brundel
Registered
Although it appears most ADHD drugs work by increasing dopamine they do not do so in the same way Parkinsons drugs (LDOPA)
do.
Part of the problem is that anti-parkinson drugs are designed to boost dopamine levels in a different region of the brain from the region where such deficits result in ADHD.
The thing is, anti-parkinson drugs work by boosting levels of the precursors to dopamine, the chemicals that the body uses to put dopamine together, essentially. Using these drugs can increase the amount of dopamine available within the cell, which is referred to as vesicular dopamine, because it is stored in little baggies called vesicles within the brain cells. Vesicular dopamine is then available for the cell to send out into the region between brain cells, called the synapse, where it acts as a chemical signal to a nearby cell. When dopamine exists the presynaptic cell into the synapse, it can be referred to as synaptic dopamine. Synaptic dopamine levels can be influenced by vesicular dopamine levels in some situations, and apparently many researchers believe that low synaptic dopamine levels in parkinson's patients are a result of low vesicular dopamine levels, and thus it makes sense to give them L-dopa as a means of boosting production of dopamine within certain brain cells.
By contrast, while ADHD does appear to result from, or at least involve low synaptic dopamine levels in certain striatal, prefrontal, and frontal regions of the brain, the majority of research appears to show that this is not influenced by vesicular dopamine levels. The presynaptic cells have plenty of dopamine, in other words, and the reason for the lack of dopamine in the synapse is due to something else, in this case the excess of dopamine transport proteins on the cell membrane, which remove the dopamine from the synapse. Too many transport proteins (I've seen estimates around 70% higher in ADHD persons) means that too much dopamine is removed from the synapse too quickly, and thus boosting the amount within the cell is not going to help much.
Most ADHD medications affect dopamine transporters, along with transport proteins for a related chemical called norepinephrine. Norepinephrine has also been implicated as being involved in ADHD, and additionally norepinephrine transport proteins may also be capable of removing dopamine from the synapse.
This is why one would use Adderall on an ADHD patient (causes transport proteins to work in reverse), but not on a Parkinson's patient, and why L-Dopa would work on a Parkinson's patient and not on an ADHD patient.
May not want to ditch the Adderall in exchange for Prolactrone.
do.
Part of the problem is that anti-parkinson drugs are designed to boost dopamine levels in a different region of the brain from the region where such deficits result in ADHD.
The thing is, anti-parkinson drugs work by boosting levels of the precursors to dopamine, the chemicals that the body uses to put dopamine together, essentially. Using these drugs can increase the amount of dopamine available within the cell, which is referred to as vesicular dopamine, because it is stored in little baggies called vesicles within the brain cells. Vesicular dopamine is then available for the cell to send out into the region between brain cells, called the synapse, where it acts as a chemical signal to a nearby cell. When dopamine exists the presynaptic cell into the synapse, it can be referred to as synaptic dopamine. Synaptic dopamine levels can be influenced by vesicular dopamine levels in some situations, and apparently many researchers believe that low synaptic dopamine levels in parkinson's patients are a result of low vesicular dopamine levels, and thus it makes sense to give them L-dopa as a means of boosting production of dopamine within certain brain cells.
By contrast, while ADHD does appear to result from, or at least involve low synaptic dopamine levels in certain striatal, prefrontal, and frontal regions of the brain, the majority of research appears to show that this is not influenced by vesicular dopamine levels. The presynaptic cells have plenty of dopamine, in other words, and the reason for the lack of dopamine in the synapse is due to something else, in this case the excess of dopamine transport proteins on the cell membrane, which remove the dopamine from the synapse. Too many transport proteins (I've seen estimates around 70% higher in ADHD persons) means that too much dopamine is removed from the synapse too quickly, and thus boosting the amount within the cell is not going to help much.
Most ADHD medications affect dopamine transporters, along with transport proteins for a related chemical called norepinephrine. Norepinephrine has also been implicated as being involved in ADHD, and additionally norepinephrine transport proteins may also be capable of removing dopamine from the synapse.
This is why one would use Adderall on an ADHD patient (causes transport proteins to work in reverse), but not on a Parkinson's patient, and why L-Dopa would work on a Parkinson's patient and not on an ADHD patient.
May not want to ditch the Adderall in exchange for Prolactrone.
these babies are in stock at orbitnutrition!
brundel
Registered
Awesome!
packers6211
Registered
Sweeet it's arrived!! Let's see how long it stays in stock hehehe.
5thou
Registered
Already got mine. Thanks!
brundel
Registered
You guys are really gonna dig this stuff. Everyone whos taken it is giving super positive feedback.
