Some good novice cycles in my mind
1-12 Test C or E 400-500 mg/w
1-12 Deca Durabolin 400 mg/w
8-12 Dianabol 30 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
1-12 Test C or E 400-500 mg/w
1-12 Equipoise 400 mg/w
8-12 Stanozolol 30 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
1-12 Test C or E 400-500 mg/w
1-12 Primobolan 500 mg/w
8-12 Anavar 40-50 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
note: primobolan is probably the most faked AAS
1-12 Sustanon 500 mg/w
1-12 Parabolan 228 mg/w
8-12 Stanozolol 30 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
Note: Parabolin is difficult to obtain but is the only trenbolone developed for human use and tends to give less side effects
Rational for oral timing: Gains tend to diminish at or around week 8. This is likely for a miriad of reasons one being an increase in myostatin expression. C17 methyl alkylated compounds, most orals, do not bind tightly to the androgen receptor. Rather, they tend to act through modulation of glucocorticoid pathways and are thus thought to be anti-catabolic rather than anabolic. This is a rather general statement of course. Still, the idea is that by introduction of one of these compounds at the plateau point in a cycle allows another pathway to become active and thus gains are more continuous.
Rational for testosterone in all cycles: Test is required for many functions in the male body. Briefly, these include activity in the lymbic system, sexual function, conversion to estrogen which is important for bone and joint health, and many others. failure to use it often leads to sexual dysfunction, joint pain and brain fog.
Rational for use of an additional anabolic: Anabolic steroids were developed to eliminate or limit the negative aspects of testosterones such as aromatization to estrogens and androgenic effects such as increased body hair growth and prostate enlargement. So, rather than use 2x the amount of testosterone in a cycle and be forced to deal with added side effects and the requirement for additional ancillary compounds the idea here is to use compounds that will not add to those problems but add anabolism.
Rational for use of Adex: Many users will have estrogenic problems even at 400-500 mg/w of testosterone. In the TRT setting some even have problems on 100 mg/w. Adex is a second generation type 1 aromatase inhibitor and is generally well tolerated. Roughly 0.5 mg every other day tends to reduce estrogen in the average male body by half, which may roughly place estrogen close to or in the normal range. Of course blood studies are required to dial this in.
PCT rational: Clomid was developed to act on the arcuate nucleus cells in the hypothalamus by blocking estrogen receptors, which under normal circumstances inhibits the secretion of GNRH, a protein hormone required to stimulate release of FSH and LH from the pituitary. These later 2 protein hormones then stimulate the tested to produce sperm and testosterone respectively among other things. Clomid is better than some SERMs for this purpose as it was selected for this purpose more or less (actually developed to stimulate the same cells in the female and thus ovulation). After approximately 2-3 weeks clomifene looses some of its effectiveness. Here is where Nolvadex is useful. It also binds to the arcuate nucleus cells (as well as ER in breast and uterine tissues) and has been shown to restore GnRH signaling under reduced clomifene response. Use of the two together thus should be more effective and as an added bonus will guard against estrogen dominance which can occur during PCT and lead to gynocomastia.
1-12 Test C or E 400-500 mg/w
1-12 Deca Durabolin 400 mg/w
8-12 Dianabol 30 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
1-12 Test C or E 400-500 mg/w
1-12 Equipoise 400 mg/w
8-12 Stanozolol 30 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
1-12 Test C or E 400-500 mg/w
1-12 Primobolan 500 mg/w
8-12 Anavar 40-50 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
note: primobolan is probably the most faked AAS
1-12 Sustanon 500 mg/w
1-12 Parabolan 228 mg/w
8-12 Stanozolol 30 mg/d
1-14 Adex 0.5 mg EOD
14-18 Clomid 100/50/50/50/-/-
14-18 Nolvadex -/-/20/20/20/20
Note: Parabolin is difficult to obtain but is the only trenbolone developed for human use and tends to give less side effects
Rational for oral timing: Gains tend to diminish at or around week 8. This is likely for a miriad of reasons one being an increase in myostatin expression. C17 methyl alkylated compounds, most orals, do not bind tightly to the androgen receptor. Rather, they tend to act through modulation of glucocorticoid pathways and are thus thought to be anti-catabolic rather than anabolic. This is a rather general statement of course. Still, the idea is that by introduction of one of these compounds at the plateau point in a cycle allows another pathway to become active and thus gains are more continuous.
Rational for testosterone in all cycles: Test is required for many functions in the male body. Briefly, these include activity in the lymbic system, sexual function, conversion to estrogen which is important for bone and joint health, and many others. failure to use it often leads to sexual dysfunction, joint pain and brain fog.
Rational for use of an additional anabolic: Anabolic steroids were developed to eliminate or limit the negative aspects of testosterones such as aromatization to estrogens and androgenic effects such as increased body hair growth and prostate enlargement. So, rather than use 2x the amount of testosterone in a cycle and be forced to deal with added side effects and the requirement for additional ancillary compounds the idea here is to use compounds that will not add to those problems but add anabolism.
Rational for use of Adex: Many users will have estrogenic problems even at 400-500 mg/w of testosterone. In the TRT setting some even have problems on 100 mg/w. Adex is a second generation type 1 aromatase inhibitor and is generally well tolerated. Roughly 0.5 mg every other day tends to reduce estrogen in the average male body by half, which may roughly place estrogen close to or in the normal range. Of course blood studies are required to dial this in.
PCT rational: Clomid was developed to act on the arcuate nucleus cells in the hypothalamus by blocking estrogen receptors, which under normal circumstances inhibits the secretion of GNRH, a protein hormone required to stimulate release of FSH and LH from the pituitary. These later 2 protein hormones then stimulate the tested to produce sperm and testosterone respectively among other things. Clomid is better than some SERMs for this purpose as it was selected for this purpose more or less (actually developed to stimulate the same cells in the female and thus ovulation). After approximately 2-3 weeks clomifene looses some of its effectiveness. Here is where Nolvadex is useful. It also binds to the arcuate nucleus cells (as well as ER in breast and uterine tissues) and has been shown to restore GnRH signaling under reduced clomifene response. Use of the two together thus should be more effective and as an added bonus will guard against estrogen dominance which can occur during PCT and lead to gynocomastia.
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