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Clomid is a SERM that was originally intended for females to treat infertility. It was later used by men to treat impotence.
When used in pct (or when exogenous hormones are abscent), it binds to the estrogen receptors in hypothalamus and anterior pituitary without activating them and inhibits the negative feedback. This will stimulate the body to produce LH and FSH to simulate endo test production.
Now if the leydig cells have been dormant for a long time, it might be harder to reverse testicular hypertrophy, that's when hcg is used.
So in conclusion, it may help reverse testicle shrinkage but it all depends on the response from your leydig cells to GnRH.
okat thnx bro im just tryin to figure out how long i should run clomid for i ve been doin 50 for a week and now 25 eod the sides were killlin me but my balls are still big
Clomid support gonadotropin release and it wont prevent testo supression on cycle . Actually there is merit to using hCG throughout the use of AAS, so the HPTA is less suppressed and atrophied (testes). This could make recovery faster. If you avoid atrophy of the testes, recovery of your natural testosterone should be much faster. The added endogenous testosterone may help muscle mass to some extent, though its effects are probably going to be insignificant next to the exogenous AAS.
I think there is some merit to a shorter PCT, or even just the use of clomid, if the cycle has been brief. I think the impact PCT is going to have will depend a lot on how suppressed your HPT axis is (usually related to the length of use).