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Serious problems with my knees

KelJu

Thats Dr. Keke to you!
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I finally got my legs back to somewhat respectable status, and now my knees are fucking up again, both of them. They hurt in the exact same spot on both sides. My right knee started first, and a week later, my left started to hurt.

I am not even truly certain that the problem is my knee. It feels like tendentious of the elbow, except in the back of my legs. I haven't changed anything in my training. For both legs to develop the same problem at the same time is fucking with my head. I can;t even guess at what is causing it.
 
Last edited:
It could be any number of things.

Are you on gear?
 
Never mind...
 
Last edited:
Yes. Why?

Can you post your cycle?

gear affects collagen synthesis, some increase some decrease. When I get home, iml send you an article on gear and collagen synthesis that you may be interested in.
 
Cut Mix 150, Dragon Pharma
Substance: tren a, test prop, masteron
300mg eod

Anastrozole
2mg every day for the next two weeks
 
Cut Mix 150, Dragon Pharma
Substance: tren a, test prop, masteron
300mg eod

Anastrozole
2mg every day for the next two weeks

thats way too high for adex imo. and you dont need to take it ed, it has a longer half than aromasin and you can take it eod. I'd cut back to 1mg eod.

Any specific reason on why you're taking such a high dose of ai?

here's the article I was talking about:


How to increase collegen synthesis!! (i.e. - strengthen those tendons and ligiments)
originally posted by AnimalMass on competitivemuscle.com

While injecting
test increases protein syntesis by roughly 50 times, depending on dose and time, most bodybuilders forget that it will reduce collagen synthesis by more than 50% -- more like 80%, giving you the collagen synthesis rate of a senior citizen. Since collagen makes up tendons, bros are very prone to injury if they continue to lift very heavy, unless they cycle off T and let their collagen synthesis get back to normal. It's like having the skeletal muscle of a gorilla with the tendons of a very old man.

Winstrol increases collagen synthesis. It will give you bigger tendons. However, your body compensates for this by making them more brittle, weaker, and more prone to injury. I can't tell you how many bros work out anaerobically and become injured while on winstrol. Guys who lift in the 1-5 rep range while on winstrol, to baseball players who sprint all out from a stationary position -- winstrol should be the LAST drug they choose. Most of them like winstrol because they don't get the weight gain from it but it is very detrimental to bros who train for any sport anaerobically. Tendons tear easily on it.


Also, the drugs I mention increase collagen syn while also increasing collagen cross-linking integrity, making for a much stronger tendon.


Winstrol, on the other hand, will dramatically increase collagen syn, but ironically it decreases collagen cross-linking integrity, thus making a much weaker tendon.


You can plan a cycle of AAS which will increase collagen synthesis and skeletal muscle growth at the same time. The key is the drug(s) you choose.


deca
, Equipoise, Anavar, and primobolan will ALL increase skeletal muscle while at the same time dramatically increase collagen syn and bone mass and density, leaving you with a substantially reduced chance of becoming injured than if you choose to use AAS like sus, cyp, or enth.

While testosterone will increase bone mass and density, even at supra-physiological levels, the result is weaker tendons due to inhibition of collagen syn.


To plan a cycle where the goal is to increase skeletal muscle mass/strength while at the same time increase joint/tendon/ligament strength, enough to keep up with the dramatic increase in skeletal muscle, you must choose drugs like Eq,
deca, Anavar, or Primo as the base of your cycle. Testosterone and its esters can be added to your cycle to keep levels within a 'normal' physiological range (ie, 100-200 mg/wk) but must not go above this. Since drugs like eq, deca, anavar and primo will reduce endogenous, natural levels of test, these levels may be maintained with exogenous test in the 100-200 mg/wk range. test at this dose will not inhibit collagen syn, but paradoxically, will help increase it. It is when exogenous testosterone is used > 200 mg/wk that collagen syn is inhibited.

deca
@ 3 mg/kg a week(about 270 mg/wk for a 200 lb male) will increase procollagen III levels by 270% by week 2. Procollagen III is a primary indicator used to determine the rate of collagen syn. As you can see, deca is a very good drug at giving you everything you want -- an increase in collagen syn, an increase in skeletal muscle, and increases in bone mass and density. The one thing it does not give you is wood

primobolan
, @ 5 mg/kg, will increase collagen synthesis by roughly 180% -- less than deca and equipoise but still substantial.

Equipoise @ 3 mg/kg will increase procollagen III by approximately 340% -- slightly better than
deca.

Oxandrolone has over a hundred studies documenting its effectiveness at treating patients needing rapid increases in collagen syn to enhance healing.


These drugs have longer half-lives than most other AAS, so this should be considered when timing your post cycle
clomid use. Here they are:

deca
: 15 days Equipoise: 14 days primobolan: 10.5 days

Anavar has a half-life of only 8 hours so it should not pose a problem.


GH is probably the most remarkable drug at increasing collagen synthesis. It increases collagen syn in a dose dependant manner -- the more you use, the more you will increase collagen syn. It has also demonstrated this ability in short and long term studies. From what I've read,
HGH at 6 iu/day increased the collagen deposition rate by around 250% in damaged collagen structures. This result indicates that the increased biomechanical strength of wounds to collagen structures treated with biosynthetic human growth hormone was produced by an increased deposition of collagen in the collagen structures.

Eq, primo, anavar, and
deca are all good -- they increase several biomakers of collagen syn -- ie, type III, II, I, procollagen markers. GH just seems to do so most dramatically.

Use of any of these drugs @ supra-physiological levels with a maintenance dose of
test will increase collagen syn while at the same time increase skeletal muscle mass. Skeletal muscle mass gains will not be as dramatic as with large testosterone doses but you have to weigh the risk/reward basis for yourself. Also, these drugs do not satisfy the libido like testosterone, but that is not the point of this thread. It is only to demonstrate that you can increase skeletal muscle and collagen syn at the same time with certain AAS -- the decision is up to you.

AnimalMass
 
thats way too high for adex imo. and you dont need to take it ed, it has a longer half than aromasin and you can take it eod. I'd cut back to 1mg eod.

Any specific reason on why you're taking such a high dose of ai?

here's the article I was talking about:


How to increase collegen synthesis!! (i.e. - strengthen those tendons and ligiments)
originally posted by AnimalMass on competitivemuscle.com

While injecting
test increases protein syntesis by roughly 50 times, depending on dose and time, most bodybuilders forget that it will reduce collagen synthesis by more than 50% -- more like 80%, giving you the collagen synthesis rate of a senior citizen. Since collagen makes up tendons, bros are very prone to injury if they continue to lift very heavy, unless they cycle off T and let their collagen synthesis get back to normal. It's like having the skeletal muscle of a gorilla with the tendons of a very old man.

Winstrol increases collagen synthesis. It will give you bigger tendons. However, your body compensates for this by making them more brittle, weaker, and more prone to injury. I can't tell you how many bros work out anaerobically and become injured while on winstrol. Guys who lift in the 1-5 rep range while on winstrol, to baseball players who sprint all out from a stationary position -- winstrol should be the LAST drug they choose. Most of them like winstrol because they don't get the weight gain from it but it is very detrimental to bros who train for any sport anaerobically. Tendons tear easily on it.


Also, the drugs I mention increase collagen syn while also increasing collagen cross-linking integrity, making for a much stronger tendon.


Winstrol, on the other hand, will dramatically increase collagen syn, but ironically it decreases collagen cross-linking integrity, thus making a much weaker tendon.


You can plan a cycle of AAS which will increase collagen synthesis and skeletal muscle growth at the same time. The key is the drug(s) you choose.


deca
, Equipoise, Anavar, and primobolan will ALL increase skeletal muscle while at the same time dramatically increase collagen syn and bone mass and density, leaving you with a substantially reduced chance of becoming injured than if you choose to use AAS like sus, cyp, or enth.

While testosterone will increase bone mass and density, even at supra-physiological levels, the result is weaker tendons due to inhibition of collagen syn.


To plan a cycle where the goal is to increase skeletal muscle mass/strength while at the same time increase joint/tendon/ligament strength, enough to keep up with the dramatic increase in skeletal muscle, you must choose drugs like Eq,
deca, Anavar, or Primo as the base of your cycle. Testosterone and its esters can be added to your cycle to keep levels within a 'normal' physiological range (ie, 100-200 mg/wk) but must not go above this. Since drugs like eq, deca, anavar and primo will reduce endogenous, natural levels of test, these levels may be maintained with exogenous test in the 100-200 mg/wk range. test at this dose will not inhibit collagen syn, but paradoxically, will help increase it. It is when exogenous testosterone is used > 200 mg/wk that collagen syn is inhibited.

deca
@ 3 mg/kg a week(about 270 mg/wk for a 200 lb male) will increase procollagen III levels by 270% by week 2. Procollagen III is a primary indicator used to determine the rate of collagen syn. As you can see, deca is a very good drug at giving you everything you want -- an increase in collagen syn, an increase in skeletal muscle, and increases in bone mass and density. The one thing it does not give you is wood

primobolan
, @ 5 mg/kg, will increase collagen synthesis by roughly 180% -- less than deca and equipoise but still substantial.

Equipoise @ 3 mg/kg will increase procollagen III by approximately 340% -- slightly better than
deca.

Oxandrolone has over a hundred studies documenting its effectiveness at treating patients needing rapid increases in collagen syn to enhance healing.


These drugs have longer half-lives than most other AAS, so this should be considered when timing your post cycle
clomid use. Here they are:

deca
: 15 days Equipoise: 14 days primobolan: 10.5 days

Anavar has a half-life of only 8 hours so it should not pose a problem.


GH is probably the most remarkable drug at increasing collagen synthesis. It increases collagen syn in a dose dependant manner -- the more you use, the more you will increase collagen syn. It has also demonstrated this ability in short and long term studies. From what I've read,
HGH at 6 iu/day increased the collagen deposition rate by around 250% in damaged collagen structures. This result indicates that the increased biomechanical strength of wounds to collagen structures treated with biosynthetic human growth hormone was produced by an increased deposition of collagen in the collagen structures.

Eq, primo, anavar, and
deca are all good -- they increase several biomakers of collagen syn -- ie, type III, II, I, procollagen markers. GH just seems to do so most dramatically.

Use of any of these drugs @ supra-physiological levels with a maintenance dose of
test will increase collagen syn while at the same time increase skeletal muscle mass. Skeletal muscle mass gains will not be as dramatic as with large testosterone doses but you have to weigh the risk/reward basis for yourself. Also, these drugs do not satisfy the libido like testosterone, but that is not the point of this thread. It is only to demonstrate that you can increase skeletal muscle and collagen syn at the same time with certain AAS -- the decision is up to you.

AnimalMass



I doubled the dose of aromasin because my balls were hurting. I didn't think that an increase would cause any problems. I thought about trying deca, but just never got around it. Maybe that is the solution in the future. It's too late now. I basically can't even fucking workout my legs until they heal or I have surgery. I don't even have insurance at the moment, so I guess I'm proper fucked.
 
This is not just a product plug, we have had many great reviews from people with tendonitis and other joint issues that use our Flex Rx and have great results: Flex Rx.

One of the bodybuilders that swears by this product is Tracy Bodner (aka IslandGirl).
Feel free to ask her in her Q & A thread: http://www.ironmagazineforums.com/e...8-q-ifbb-pro-tracy-bodner-aka-islandgirl.html

Another thing that can help with joint issues is fish oil, we are actually bringing back our Essence EFA product except it's a new formula and enteric coated gel caps.
 
Muscle Gelz Transdermals
IronMag Labs Prohormones
^^^Good looking product Prince. I may have to try some out.
 
I finally got my legs back to somewhat respectable status, and now my knees are fucking up again, both of them. They hurt in the exact same spot on both sides. My right knee started first, and a week later, my left started to hurt.

I am not even truly certain that the problem is my knee. It feels like tendentious of the elbow, except in the back of my legs. I haven't changed anything in my training. For both legs to develop the same problem at the same time is fucking with my head. I can;t even guess at what is causing it.

The area you noted is an attachment site for several muscles. Semitendonosis, semimembranosis (both hamstring muscles), gracilis and sartorius. The problem may or may not be muscular.

Have you been doing any new kinds of exercise?
How deep does the pain feel?
How would you best describe the pain?
Do any specific actions or activities aggravate the problem?
Have you ever had pain to that specific area before?
Does the area feel inflamed (puffy) or warm to the touch (indicative of inflammation).
Does it hurt if you press on it?
 
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